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Items 1 to 10 of about 1340617
2. Andresen M, Díaz O, Troncoso S: [Nitric oxide in respiratory diseases]. Rev Med Chil; 1997 Aug;125(8):935-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Nitric oxide in respiratory diseases].
  • Recent reports have described a pathogenic role of nitric oxide in several respiratory disease.
  • It is specially useful in the adult respiratory distress syndrome, where it acts as a selective vasodilator and improves gas exchange, decreasing pulmonary shunting.
  • This article review the metabolism, mechanisms of action, potential uses and adverse effects of nitric oxide in respiratory disease.
  • [MeSH-major] Asthma / drug therapy. Bronchodilator Agents / therapeutic use. Nitric Oxide / therapeutic use. Respiratory Distress Syndrome, Newborn / drug therapy. Vasodilator Agents / therapeutic use

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  • (PMID = 9567400.001).
  • [ISSN] 0034-9887
  • [Journal-full-title] Revista médica de Chile
  • [ISO-abbreviation] Rev Med Chil
  • [Language] spa
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] CHILE
  • [Chemical-registry-number] 0 / Bronchodilator Agents; 0 / Vasodilator Agents; 31C4KY9ESH / Nitric Oxide
  • [Number-of-references] 25
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3. Christou H, Van Marter LJ, Wessel DL, Allred EN, Kane JW, Thompson JE, Stark AR, Kourembanas S: Inhaled nitric oxide reduces the need for extracorporeal membrane oxygenation in infants with persistent pulmonary hypertension of the newborn. Crit Care Med; 2000 Nov;28(11):3722-7
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  • [Title] Inhaled nitric oxide reduces the need for extracorporeal membrane oxygenation in infants with persistent pulmonary hypertension of the newborn.
  • OBJECTIVE: We previously reported improved oxygenation, but no change, in rates of extracorporeal membrane oxygenation (ECMO) use or death among infants with persistent pulmonary hypertension of the newborn who received inhaled nitric oxide (NO) with conventional ventilation, irrespective of lung disease.
  • The goal of our study was to determine whether treatment with inhaled NO improves oxygenation and clinical outcomes in infants with persistent pulmonary hypertension of the newborn and associated lung disease who are ventilated with high-frequency oscillatory ventilation (HFOV).
  • PATIENTS: We studied infants with a gestational age of > or =34 wks who were receiving mechanical ventilatory support and had echocardiographic and clinical evidence of pulmonary hypertension and hypoxemia (PaO2 < or =100 mm Hg on FIO2 = 1.0), despite optimal medical management Infants with congenital heart disease, diaphragmatic hernia, or other major anomalies were excluded.
  • We speculate that HFOV enhances the effectiveness of inhaled NO treatment in infants with persistent pulmonary hypertension of the newborn and associated lung disease.
  • [MeSH-major] Extracorporeal Membrane Oxygenation. Nitric Oxide / administration & dosage. Persistent Fetal Circulation Syndrome / therapy

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  • (PMID = 11098980.001).
  • [ISSN] 0090-3493
  • [Journal-full-title] Critical care medicine
  • [ISO-abbreviation] Crit. Care Med.
  • [Language] eng
  • [Grant] United States / FDA HHS / FD / FD-R00133136; United States / NHLBI NIH HHS / HL / P50 HL46491; United States / NHLBI NIH HHS / HL / R01 HL55454; etc
  • [Publication-type] Clinical Trial; Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] UNITED STATES
  • [Chemical-registry-number] 31C4KY9ESH / Nitric Oxide; S88TT14065 / Oxygen
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4. Kinsella JP, Neish SR, Ivy DD, Shaffer E, Abman SH: Clinical responses to prolonged treatment of persistent pulmonary hypertension of the newborn with low doses of inhaled nitric oxide. J Pediatr; 1993 Jul;123(1):103-8
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  • [Title] Clinical responses to prolonged treatment of persistent pulmonary hypertension of the newborn with low doses of inhaled nitric oxide.
  • We studied the efficacy of low-dose nitric oxide inhalation in nine consecutive patients with severe persistent pulmonary hypertension of the newborn (PPHN) who were candidates for extracorporeal membrane oxygenation (ECMO).
  • All patients had marked hypoxemia despite aggressive ventilator management and echocardiographic evidence of pulmonary hypertension.
  • Infants were initially treated with inhaled nitric oxide at 20 ppm for 4 hours and then at 6 ppm for 20 hours.
  • Sustained improvement in oxygenation was achieved in eight patients treated with inhaled nitric oxide for 24 hours at 6 ppm (arterial/alveolar oxygen ratio, 0.270 +/- 0.053 at 24 hours; p < 0.001 vs baseline).
  • One patient with overwhelming sepsis had an initial improvement of oxygenation with nitric oxide but required ECMO for multiorgan and cardiac dysfunction.
  • We conclude that low doses of nitric oxide cause sustained clinical improvement in severe PPHN and may reduce the need for ECMO.
  • [MeSH-major] Nitric Oxide / administration & dosage. Persistent Fetal Circulation Syndrome / drug therapy. Vasodilator Agents / administration & dosage

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  • [CommentIn] J Pediatr. 1993 Jul;123(1):76-9 [8320629.001]
  • (PMID = 8320602.001).
  • [ISSN] 0022-3476
  • [Journal-full-title] The Journal of pediatrics
  • [ISO-abbreviation] J. Pediatr.
  • [Language] eng
  • [Grant] United States / NHLBI NIH HHS / HL / HL-01932; United States / NHLBI NIH HHS / HL / HL-41012; United States / NHLBI NIH HHS / HL / HL-46481
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] UNITED STATES
  • [Chemical-registry-number] 0 / Vasodilator Agents; 31C4KY9ESH / Nitric Oxide; S88TT14065 / Oxygen
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5. Ciebiada M, Cichocki P, Kasztalska K, Majewski S, Gorska-Ciebiada M, Gorski P: Orally exhaled nitric oxide in patients with seasonal allergic rhinitis during natural pollen season. Am J Rhinol Allergy; 2012 Jan-Feb;26(1):e32-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Orally exhaled nitric oxide in patients with seasonal allergic rhinitis during natural pollen season.
  • BACKGROUND: Nitric oxide (NO) is the endogenous molecule involved in regulatory, protective, and defensive mechanisms.
  • [MeSH-major] Asthma / diagnosis. Breath Tests. Nitric Oxide / metabolism. Rhinitis, Allergic, Seasonal / diagnosis

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  • (PMID = 22391078.001).
  • [ISSN] 1945-8932
  • [Journal-full-title] American journal of rhinology & allergy
  • [ISO-abbreviation] Am J Rhinol Allergy
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Allergens; 31C4KY9ESH / Nitric Oxide; 37341-29-0 / Immunoglobulin E
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6. ben Anes A, Fetoui H, Bchir S, ben Nasr H, Chahdoura H, Chabchoub E, Yacoub S, Garrouch A, Benzarti M, Tabka Z, Chahed K: Increased oxidative stress and altered levels of nitric oxide and peroxynitrite in Tunisian patients with chronic obstructive pulmonary disease: correlation with disease severity and airflow obstruction. Biol Trace Elem Res; 2014 Oct;161(1):20-31
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  • [Title] Increased oxidative stress and altered levels of nitric oxide and peroxynitrite in Tunisian patients with chronic obstructive pulmonary disease: correlation with disease severity and airflow obstruction.
  • This study was aimed to evaluate the oxidant-antioxidant imbalance in the pathogenesis of chronic obstructive pulmonary disease (COPD) in Tunisians.
  • We also evaluated the level of nitric oxide (NO) and peroxynitrite in plasma from COPD patients and healthy controls.
  • Pulmonary functional tests were performed by body plethysmography.
  • [MeSH-major] Nitric Oxide / blood. Oxidative Stress. Peroxynitrous Acid / blood. Pulmonary Disease, Chronic Obstructive / blood

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  • (PMID = 25074430.001).
  • [ISSN] 1559-0720
  • [Journal-full-title] Biological trace element research
  • [ISO-abbreviation] Biol Trace Elem Res
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Advanced Oxidation Protein Products; 0 / Thiobarbituric Acid Reactive Substances; 14691-52-2 / Peroxynitrous Acid; 31C4KY9ESH / Nitric Oxide; 4Y8F71G49Q / Malondialdehyde; EC 1.11.1.6 / Catalase; EC 1.11.1.9 / Glutathione Peroxidase; EC 1.15.1.1 / Superoxide Dismutase; GAN16C9B8O / Glutathione
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7. Zhang Y, Boddicker KA, Rhee BJ, Davies LR, Kerber RE: Effect of nitric oxide synthase modulation on resuscitation success in a swine ventricular fibrillation cardiac arrest model. Resuscitation; 2005 Oct;67(1):127-34
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  • [Title] Effect of nitric oxide synthase modulation on resuscitation success in a swine ventricular fibrillation cardiac arrest model.
  • BACKGROUND: We have demonstrated previously that the nitric oxide synthase (NOS) inhibitor N(G)-nitro-L-arginine (L-NNA) decreases free radical generation and nitrosative injury via peroxynitrite formation after epicardial dc shocks.
  • We used the non-selective NOS inhibitor L-NNA and the selective neuronal NOS inhibitor ARR-17477, the NOS donor S-nitroso-N-acetylpenicillamine (SNAP) and the vasodilator Enalaprilat, which lowers arterial pressure via a non-NO mechanism.
  • Part II: 35 pigs undergoing 6-8 min VF were randomized to three groups: a no-L-NNA group (n=13) receiving IV saline, an L-NNA group (n=11) receiving IV L-NNA (5 mg/kg) and an ARR17477 group (n=11) receiving IV ARR17477 (5 mg/kg) before VF.
  • All animals in Part II underwent unsupported VF (no chest compression or ventilation) for 6 min (n=13) or 8 min (n=22); closed-chest compression, ventilation and epinephrine (adrenaline) were employed after defibrillation.
  • In Part II, there was no significant difference between L-NNA, ARR17477 and control pigs in ROSC.
  • [MeSH-major] Electric Countershock / methods. Heart Arrest / therapy. Nitric Oxide Synthase / drug effects. Nitroarginine / pharmacology. Ventricular Fibrillation / therapy

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  • (PMID = 16039037.001).
  • [ISSN] 0300-9572
  • [Journal-full-title] Resuscitation
  • [ISO-abbreviation] Resuscitation
  • [Language] eng
  • [Grant] United States / NHLBI NIH HHS / HL / HL71676-01
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] Ireland
  • [Chemical-registry-number] 0 / Enzyme Inhibitors; 2149-70-4 / Nitroarginine; EC 1.14.13.39 / Nitric Oxide Synthase
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8. Kimura H, Miura S, Shigematsu T, Ohkubo N, Tsuzuki Y, Kurose I, Higuchi H, Akiba Y, Hokari R, Hirokawa M, Serizawa H, Ishii H: Increased nitric oxide production and inducible nitric oxide synthase activity in colonic mucosa of patients with active ulcerative colitis and Crohn's disease. Dig Dis Sci; 1997 May;42(5):1047-54
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  • [Title] Increased nitric oxide production and inducible nitric oxide synthase activity in colonic mucosa of patients with active ulcerative colitis and Crohn's disease.
  • It is postulated that an enhanced production of nitric oxide by inflamed intestine plays a role in the pathophysiology of active inflammatory bowel disease.
  • In this study, systemic NOx concentrations and colonic nitric oxide synthase activity were determined in patients with ulcerative colitis or Crohn's disease.
  • The relationship between these two parameters and disease activity, as well as differences in nitric oxide synthase activity between ulcerative colitis and Crohn's disease, were areas of specific focus.
  • Patients with active ulcerative colitis and Crohn's disease had significantly elevated plasma NOx concentrations; a positive correlation was found between NOx values and inducible nitric oxide synthase activities in the active mucosa of these patients.
  • In active ulcerative colitis, levels of inducible nitric oxide synthase were significantly elevated in both normal and inflamed mucosa, although inducible nitric oxide synthase activity was higher in the latter.
  • These colonic inducible nitric oxide synthase activities correlated well with the results of endoscopic and histologic grading of inflammation.
  • There was no increase in constitutive nitric oxide synthase activity in patients with active ulcerative colitis.
  • However, constitutive nitric oxide synthase activity was significantly increased in the inflamed mucosa in patients with Crohn's disease.
  • In Crohn's disease, elevated inducible nitric oxide synthase activity was found in both normal and inflamed mucosa, with no significant difference between the tissues.
  • Such differences in nitric oxide production in the colonic mucosa possibly reflect the significant differences in the pathophysiology and characteristic clinical features between ulcerative colitis and Crohn's disease.
  • [MeSH-major] Colitis, Ulcerative / metabolism. Colon / metabolism. Crohn Disease / metabolism. Intestinal Mucosa / metabolism. Nitric Oxide / biosynthesis. Nitric Oxide Synthase / biosynthesis

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  • (PMID = 9149061.001).
  • [ISSN] 0163-2116
  • [Journal-full-title] Digestive diseases and sciences
  • [ISO-abbreviation] Dig. Dis. Sci.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 31C4KY9ESH / Nitric Oxide; EC 1.14.13.39 / Nitric Oxide Synthase
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9. Cannon BC, Feltes TF, Fraley JK, Grifka RG, Riddle EM, Kovalchin JP: Nitric oxide in the evaluation of congenital heart disease with pulmonary hypertension: factors related to nitric oxide response. Pediatr Cardiol; 2005 Sep-Oct;26(5):565-9
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  • [Title] Nitric oxide in the evaluation of congenital heart disease with pulmonary hypertension: factors related to nitric oxide response.
  • Inhaled nitric oxide (NO) has been used in the preoperative evaluation of patients with congenital heart disease and pulmonary hypertension.
  • The purpose of this study was to characterize responses in pulmonary vascular resistance (PVR) to oxygen and increasing doses of NO during cardiac catheterization and to determine if any related factors affect the response of the pulmonary vascular bed to NO.
  • A prospective analysis of 42 patients (median age, 3.0 years) with congenital heart disease and pulmonary hypertension who underwent NO testing was performed.
  • Changes in pulmonary artery pressure, PVR, and SVR were assessed.
  • [MeSH-major] Bronchodilator Agents / pharmacology. Heart Defects, Congenital / drug therapy. Hypertension, Pulmonary / drug therapy. Nitric Oxide / pharmacology. Oxygen / pharmacology. Vascular Resistance / drug effects
  • [MeSH-minor] Administration, Inhalation. Adolescent. Adult. Blood Pressure / drug effects. Cardiac Catheterization. Child. Child, Preschool. Dose-Response Relationship, Drug. Down Syndrome / epidemiology. Drug Therapy, Combination. Female. Humans. Infant. Male. Oxygen Consumption / drug effects. Prospective Studies. Pulmonary Artery. Risk Factors. Treatment Outcome


10. Dai A, Zhang Z, Xu Y, Niu R, Duan S: [Effects of inhalation of nitric oxide on hemodynamics, blood gas and oxygen transport function in chronic obstructive pulmonary disease]. Zhongguo Ying Yong Sheng Li Xue Za Zhi; 1997 Nov;13(4):305, 325, 332
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Effects of inhalation of nitric oxide on hemodynamics, blood gas and oxygen transport function in chronic obstructive pulmonary disease].
  • [MeSH-major] Hemodynamics. Lung Diseases, Obstructive / drug therapy. Nitric Oxide / therapeutic use
  • [MeSH-minor] Administration, Inhalation. Aged. Biological Transport, Active. Blood Gas Analysis. Female. Heart Rate. Humans. Male. Middle Aged. Oxygen / metabolism

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  • (PMID = 10322954.001).
  • [ISSN] 1000-6834
  • [Journal-full-title] Zhongguo ying yong sheng li xue za zhi = Zhongguo yingyong shenglixue zazhi = Chinese journal of applied physiology
  • [ISO-abbreviation] Zhongguo Ying Yong Sheng Li Xue Za Zhi
  • [Language] chi
  • [Publication-type] Journal Article
  • [Publication-country] CHINA
  • [Chemical-registry-number] 31C4KY9ESH / Nitric Oxide; S88TT14065 / Oxygen
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