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Items 1 to 10 of about 66742
1. Taillie LS, Poti JM: Associations of Cooking With Dietary Intake and Obesity Among Supplemental Nutrition Assistance Program Participants. Am J Prev Med; 2017 Feb;52(2S2):S151-S160

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Associations of Cooking With Dietary Intake and Obesity Among Supplemental Nutrition Assistance Program Participants.
  • Therefore, frequent cooking may be more strongly associated with improved dietary outcomes among SNAP recipients than among income-eligible non-recipients.
  • METHODS: In 2016, data from low-income adults aged 19-65 years from the National Health and Nutrition Survey 2007-2010 (N=2,578) were used to examine associations of daily home-cooked dinner and weekly fast food intake with diet intake, including calories from solid fat and added sugar and key food groups (sugar-sweetened beverages, fruit, and vegetables), and prevalence of overweight/obesity.
  • Differences in these associations for SNAP recipients versus income-eligible non-recipients were analyzed, as well as whether associations were attenuated when controlling for fast food intake.
  • RESULTS: Daily home-cooked dinners were associated with small improvements in dietary intake for SNAP recipients but not for non-recipients, including lower sugar-sweetened beverage intake (-54 kcal/day), and reduced prevalence of overweight/obesity (-6%) (p<0.05).
  • Consuming at least one fast food meal/week was associated with 9.3% and 11.6% higher overweight/obesity prevalence among SNAP recipients and non-recipients, respectively (p<0.05).

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  • [Copyright] Copyright © 2016 American Journal of Preventive Medicine. Published by Elsevier Inc. All rights reserved.
  • [Cites] Obes Rev. 2008 Nov;9(6):535-47 [18346099.001]
  • [Cites] Appetite. 1999 Feb;32(1):8-14 [9989908.001]
  • [Cites] Prev Med. 2015 Jul;76:1-7 [25847732.001]
  • [Cites] Am J Clin Nutr. 2006 Aug;84(2):274-88 [16895873.001]
  • [Cites] Public Health Nutr. 2014 May;17(5):1022-30 [24107577.001]
  • [Cites] Public Health Nutr. 2002 Dec;5(6A):889-92 [12638596.001]
  • [Cites] Soc Sci Med. 2003 Feb;56(3):617-30 [12570978.001]
  • [Cites] Can J Diet Pract Res. 2010 Fall;71(3):141-5 [20825697.001]
  • [Cites] J Acad Nutr Diet. 2015 Jun;115(6):907-18.e6 [25578928.001]
  • [Cites] J Nutr Educ Behav. 2014 Jan;46(1):85-7 [23900062.001]
  • [Cites] Soc Sci Med. 2006 Nov;63(10):2591-603 [16889881.001]
  • [Cites] BMC Public Health. 2014 Aug 28;14 :886 [25168465.001]
  • [Cites] Br J Nutr. 2009 Jul;101 Suppl 2:S73-85 [19594967.001]
  • [Cites] Health Aff (Millwood). 2014 Jun;33(6):1032-9 [24889953.001]
  • [Cites] Nutr Rev. 2012 Feb;70(2):65-79 [22300594.001]
  • [Cites] N Engl J Med. 2011 Jun 23;364(25):2392-404 [21696306.001]
  • [Cites] Public Health Nutr. 2015 Jun;18(8):1397-406 [25399031.001]
  • [Cites] Am J Clin Nutr. 1998 Oct;68(4):899-917 [9771869.001]
  • [Cites] Pediatr Obes. 2013 Aug;8(4):307-24 [23554397.001]
  • [Cites] Int J Obes (Lond). 2007 Nov;31(11):1688-95 [17579632.001]
  • [Cites] Adv Nutr. 2016 Jan 15;7(1):121-34 [26773020.001]
  • [Cites] Public Health Nutr. 2014 Jan;17(1):66-72 [23340255.001]
  • [Cites] J Am Diet Assoc. 2010 May;110(5 Suppl):S52-61 [20399299.001]
  • [Cites] Health Educ Behav. 2010 Apr;37(2):211-26 [19690290.001]
  • [Cites] JAMA. 2010 May 12;303(18):1857-8 [20460625.001]
  • [Cites] Appetite. 2016 Feb 1;97:146-54 [26654888.001]
  • [Cites] Am J Prev Med. 2014 Dec;47(6):796-802 [25245799.001]
  • (PMID = 28109417.001).
  • [ISSN] 1873-2607
  • [Journal-full-title] American journal of preventive medicine
  • [ISO-abbreviation] Am J Prev Med
  • [Language] eng
  • [Grant] United States / NICHD NIH HHS / HD / P2C HD050924; United States / NIDDK NIH HHS / DK / R01 DK098072
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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2. Hull P, Emerson JS, Quirk ME, Canedo JR, Jones JL, Vylegzhanina V, Schmidt DC, Mulvaney SA, Beech BM, Briley C, Harris C, Husaini BA: A Smartphone App for Families With Preschool-Aged Children in a Public Nutrition Program: Prototype Development and Beta-Testing. JMIR Mhealth Uhealth; 2017 Aug 02;5(8):e102

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Childhood obesity prevalence is higher among preschool children in the WIC program compared to other children, and WIC improves dietary quality among low-income children.
  • The Children Eating Well (CHEW) smartphone app was developed in English and Spanish for WIC-participating families with preschool-aged children as a home-based intervention to reinforce WIC nutrition education and help prevent childhood obesity.
  • Qualitative feedback from mothers classified as non-users pointed to several important barriers that could preclude some WIC participants from using the app at all.
  • This study adds to the growing body of research focused on the application of technology-based interventions in the WIC program to promote program retention and childhood obesity prevention.

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  • [Cites] Am J Prev Med. 2014 Apr;46(4):404-8 [24650843.001]
  • [Cites] Prev Med. 2010 Sep-Oct;51(3-4):214-21 [20558196.001]
  • [Cites] J Nutr Educ Behav. 2014 May-Jun;46(3 Suppl):S87-92 [24810002.001]
  • [Cites] Nutr Rev. 2008 Mar;66(3):123-40 [18289177.001]
  • [Cites] JMIR Mhealth Uhealth. 2015 Jun 18;3(2):e69 [26088692.001]
  • [Cites] J Health Care Poor Underserved. 2014 Nov;25(4):1542-51 [25418226.001]
  • [Cites] JAMA. 2016 Jun 7;315(21):2292-9 [27272581.001]
  • [Cites] BMC Public Health. 2010 Nov 23;10:720 [21092267.001]
  • [Cites] Health Promot Pract. 2014 Jul;15(4):548-55 [24357863.001]
  • [Cites] MMWR Morb Mortal Wkly Rep. 2016 Nov 18;65(45):1256-1260 [27855143.001]
  • [Cites] J Am Diet Assoc. 2011 May;111(5):749-55 [21515124.001]
  • [Cites] Am J Clin Nutr. 2013 Nov;98 (5):1326-34 [24047917.001]
  • [Cites] J Health Commun. 2013;18(9):1039-69 [23750972.001]
  • [Cites] J Am Diet Assoc. 2004 May;104(5):736-43 [15127057.001]
  • [Cites] Milbank Q. 2009 Mar;87(1):123-54 [19298418.001]
  • [Cites] Eval Health Prof. 2006 Mar;29(1):126-53 [16510882.001]
  • [Cites] J Nutr Educ. 2001 Mar-Apr;33(2):83-94 [12031188.001]
  • [Cites] J Nutr Educ Behav. 2014 May-Jun;46(3 Suppl):S53-8 [24809997.001]
  • [Cites] Biomed Instrum Technol. 2012 Fall;Suppl:49-56 [23039777.001]
  • [Cites] J Health Care Poor Underserved. 2015 May;26(2):335-44 [25913333.001]
  • [Cites] J Diabetes Sci Technol. 2013 May 01;7(3):612-22 [23759393.001]
  • [Cites] Health Educ Q. 1988 Winter;15(4):351-77 [3068205.001]
  • [Cites] J Med Internet Res. 2013 Dec 06;15(12):e272 [24317406.001]
  • [Cites] Am J Health Behav. 2016 Jul;40(4):461-71 [27338993.001]
  • [Cites] Int J Pediatr Obes. 2006;1(2):66-76 [17907317.001]
  • [Cites] Pediatr Blood Cancer. 2012 Jan;58(1):117-21 [22076834.001]
  • [Cites] Ann Behav Med. 2005 Aug;30(1):65-73 [16097907.001]
  • [Cites] J Nutr. 1998 Mar;128(3):548-55 [9482762.001]
  • (PMID = 28768611.001).
  • [ISSN] 2291-5222
  • [Journal-full-title] JMIR mHealth and uHealth
  • [ISO-abbreviation] JMIR Mhealth Uhealth
  • [Language] eng
  • [Grant] United States / NCATS NIH HHS / TR / UL1 TR000445; United States / NCI NIH HHS / CA / U54 CA163069; United States / NCI NIH HHS / CA / U54 CA163072; United States / NCI NIH HHS / CA / U54 CA163066; United States / NCRR NIH HHS / RR / UL1 RR024975
  • [Publication-type] Journal Article
  • [Publication-country] Canada
  • [Keywords] NOTNLM ; health education / mobile apps / pediatric obesity / public health informatics
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3. Park BY, Hong J, Park H: Neuroimaging biomarkers to associate obesity and negative emotions. Sci Rep; 2017 Aug 09;7(1):7664

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Neuroimaging biomarkers to associate obesity and negative emotions.
  • Obesity is a serious medical condition highly associated with health problems such as diabetes, hypertension, and stroke.
  • Obesity is highly associated with negative emotional states, but the relationship between obesity and emotional states in terms of neuroimaging has not been fully explored.
  • The brain activity in amygdala derived from t-fMRI might be good neuroimaging biomarker for explaining the relationship between obesity and a negative emotional state.

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  • [Cites] Neuroimage. 2013 Oct 15;80:169-89 [23684877.001]
  • [Cites] Science. 2002 Jul 19;297(5580):400-3 [12130784.001]
  • [Cites] Neuroimage. 2012 Aug 15;62(2):782-90 [21979382.001]
  • [Cites] Brain. 1998 Jan;121 ( Pt 1):47-57 [9549487.001]
  • [Cites] Hum Brain Mapp. 2010 Mar;31(3):353-64 [19662657.001]
  • [Cites] Neuroimage. 2013 Oct 15;80:62-79 [23684880.001]
  • [Cites] Neuroimage. 2013 Oct 15;80:105-24 [23668970.001]
  • [Cites] Proc Natl Acad Sci U S A. 2012 Jun 12;109(24):9599-604 [22623534.001]
  • [Cites] J Am Diet Assoc. 2008 Jan;108(1):49-56 [18155989.001]
  • [Cites] Neuroimage. 2014 Apr 15;90:449-68 [24389422.001]
  • [Cites] Front Neurosci. 2014 Nov 19;8:375 [25477778.001]
  • [Cites] J Neurosci. 2016 Jan 27;36(4):1324-35 [26818519.001]
  • [Cites] Am J Clin Nutr. 2014 Aug;100(2):524-31 [24965310.001]
  • [Cites] Curr Opin Neurobiol. 2002 Apr;12(2):169-77 [12015233.001]
  • [Cites] Neuroimage. 2005 Sep;27(3):669-76 [15993629.001]
  • [Cites] Annu Rev Neurosci. 1992;15:353-75 [1575447.001]
  • [Cites] Curr Opin Pharmacol. 2007 Dec;7(6):613-6 [18032108.001]
  • [Cites] Obes Rev. 2012 Jan;13(1):43-56 [21902800.001]
  • [Cites] Behav Brain Sci. 2012 Jun;35(3):121-43 [22617651.001]
  • [Cites] Brain. 2010 Apr;133(Pt 4):1163-72 [20375138.001]
  • [Cites] Eur Eat Disord Rev. 2011 Sep-Oct;19(5):426-37 [24081718.001]
  • [Cites] Neuroimage. 2012 Aug 15;62(2):774-81 [22248573.001]
  • [Cites] Obes Res. 2001 Nov;9(11):676-84 [11707534.001]
  • [Cites] Am J Physiol Regul Integr Comp Physiol. 2003 Apr;284(4):R965-82 [12433673.001]
  • [Cites] Int J Neuropsychopharmacol. 2013 May;16(4):857-67 [22932068.001]
  • [Cites] Neuron. 2005 Oct 20;48(2):175-87 [16242399.001]
  • [Cites] Neuroimage. 2005 Feb 1;24(3):810-21 [15652316.001]
  • [Cites] Neurology. 2011 Aug 2;77(5):461-8 [21810696.001]
  • [Cites] Neuron. 2014 Jun 4;82(5):966-80 [24908482.001]
  • [Cites] Pediatrics. 2012 Oct;130(4):e856-64 [22945407.001]
  • [Cites] Eat Behav. 2005 Dec;6(4):308-17 [16257804.001]
  • [Cites] Front Neurosci. 2015 Jul 03;9:229 [26190966.001]
  • [Cites] Lancet. 2004 Jan 10;363(9403):157-63 [14726171.001]
  • [Cites] Nature. 1996 Oct 31;383(6603):812-5 [8893004.001]
  • [Cites] Behav Neurol. 1998;11(4):233-244 [11568425.001]
  • [Cites] Neuroimage. 2002 Jan;15(1):273-89 [11771995.001]
  • [Cites] Nat Neurosci. 2008 Mar;11(3):309-17 [18278039.001]
  • [Cites] Neurology. 2013 Mar 12;80(11 Suppl 3):S76-86 [23479549.001]
  • [Cites] Neuroreport. 2014 Dec 3;25(17 ):1393-8 [25325351.001]
  • [Cites] Neuron. 1996 Nov;17(5):875-87 [8938120.001]
  • [Cites] Comput Biomed Res. 1996 Jun;29(3):162-73 [8812068.001]
  • [Cites] Trends Cogn Sci. 1999 Jan;3(1):11-21 [10234222.001]
  • [Cites] PLoS One. 2009 Jul 28;4(7):e6310 [19636426.001]
  • [Cites] Neurology. 2013 Mar 12;80(11 Suppl 3):S2-6 [23479538.001]
  • (PMID = 28794427.001).
  • [ISSN] 2045-2322
  • [Journal-full-title] Scientific reports
  • [ISO-abbreviation] Sci Rep
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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4. Valerio G, Balsamo A, Baroni MG, Brufani C, Forziato C, Grugni G, Licenziati MR, Maffeis C, Miraglia Del Giudice E, Morandi A, Pacifico L, Sartorio A, Manco M, on the behalf of the Childhood Obesity Group of the Italian Society of Pediatric Endocrinology and Diabetology: Childhood obesity classification systems and cardiometabolic risk factors: a comparison of the Italian, World Health Organization and International Obesity Task Force references. Ital J Pediatr; 2017 Feb 04;43(1):19

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Childhood obesity classification systems and cardiometabolic risk factors: a comparison of the Italian, World Health Organization and International Obesity Task Force references.
  • BACKGROUND: Body Mass Index Italian reference data are available for clinical and/or epidemiological use, but no study compared the ability of this system to classify overweight and obesity and detect subjects with clustered cardiometabolic risk factors with international standards.
  • Therefore our aim was to assess 1) the agreement among the Italian Society for Pediatric Endocrinology and Diabetology (ISPED), the World Health Organisation (WHO) and the International Obesity Task Force (IOTF) Body Mass Index cut-offs in estimating overweight or obesity in children and adolescents;.
  • Prevalence of normal-weight, overweight and obesity was determined using three classification systems: ISPED, WHO and IOTF.
  • RESULTS: ISPED and IOTF classified more subjects as normal-weight or overweight and less subjects as obese as compared to WHO (p <0.0001) in the whole sample and in groups divided by gender and age.
  • The strength of agreement between the three methods compared to each other was excellent for overweight (including obesity) definition (k > 0.900), while it differed for obesity definition, ranging from the highest agreement between ISPED and IOTF (k 0.875) to the lowest between ISPED and WHO (k 0.664).
  • WHO had the highest sensitivity, while ISPED and IOTF systems had the highest specificity, in identifying obese subjects with clustered cardiometabolic risk factors.
  • CONCLUSIONS: ISPED and IOTF systems performed similarly in assessing overweight and obesity, and were more specific in identifying obese children/adolescents with clustered cardiometabolic risk factors; on the contrary, the WHO system was more sensitive.
  • Given the seriousness of the obesity epidemic, we wonder whether the WHO system should be preferable to the national standards for clinical practice and/or obesity screening.

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  • [Cites] Pediatrics. 1999 Jun;103(6 Pt 1):1175-82 [10353925.001]
  • [Cites] Nutr Metab Cardiovasc Dis. 2014 Jul;24(7):737-43 [24656140.001]
  • [Cites] BMJ. 2000 May 6;320(7244):1240-3 [10797032.001]
  • [Cites] Int J Obes Relat Metab Disord. 2003 Sep;27(9):1121-6 [12917720.001]
  • [Cites] Am J Clin Nutr. 2004 Sep;80(3):597-603 [15321798.001]
  • [Cites] Prev Med. 2016 Jun;87:151-4 [26921658.001]
  • [Cites] Pediatrics. 2004 Aug;114(2 Suppl 4th Report):555-76 [15286277.001]
  • [Cites] PLoS One. 2013 Dec 27;8(12):e83793 [24386280.001]
  • [Cites] Int J Pediatr Obes. 2011 Oct;6(5-6):325-31 [21916784.001]
  • [Cites] Obes Res. 2005 Jun;13(6):1106-15 [15976154.001]
  • [Cites] Bull World Health Organ. 2007 Sep;85(9):660-7 [18026621.001]
  • [Cites] Eur J Clin Nutr. 2002 Feb;56(2):171-80 [11857051.001]
  • [Cites] Am J Clin Nutr. 2004 May;79(5):838-43 [15113723.001]
  • [Cites] Arch Dis Child. 1995 Jul;73(1):25-9 [7639544.001]
  • [Cites] J Endocrinol Invest. 2006 Jul-Aug;29(7):581-93 [16957405.001]
  • [Cites] Pediatrics. 2009 Sep;124 Suppl 1:S35-41 [19720666.001]
  • [Cites] ISRN Obes. 2013 Jun 27;2013:392747 [24555142.001]
  • [Cites] Vital Health Stat 11. 2002 May;(246):1-190 [12043359.001]
  • [Cites] Obes Res. 2002 Aug;10(8):838-40 [12181394.001]
  • [Cites] Pediatrics. 2009 Nov;124(5):e905-12 [19858150.001]
  • [Cites] Int J Pediatr Endocrinol. 2015;2015(1):2 [25904939.001]
  • [Cites] Obesity (Silver Spring). 2014 Feb;22(2):585-9 [23616281.001]
  • [Cites] Obes Rev. 2010 Sep;11(9):645-55 [20059704.001]
  • [Cites] Eur J Clin Nutr. 2002 Oct;56(10):973-82 [12373618.001]
  • [Cites] Pediatr Obes. 2012 Aug;7(4):284-94 [22715120.001]
  • [Cites] Obes Rev. 2011 Apr;12(4):295-300 [20492539.001]
  • [Cites] Int J Obes (Lond). 2006 Apr;30(4):590-4 [16570087.001]
  • [Cites] Public Health Nutr. 2007 Oct;10(10A):1112-20 [17903318.001]
  • [Cites] Acta Diabetol. 2016 Apr;53(2):251-60 [26070771.001]
  • [Cites] Int J Obes Relat Metab Disord. 2000 Dec;24(12):1623-7 [11126215.001]
  • [Cites] Lancet. 1993 Aug 14;342(8868):418-21 [8101908.001]
  • [Cites] J Clin Epidemiol. 2003 May;56(5):456-62 [12812820.001]
  • (PMID = 28257654.001).
  • [ISSN] 1824-7288
  • [Journal-full-title] Italian journal of pediatrics
  • [ISO-abbreviation] Ital J Pediatr
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Keywords] NOTNLM ; Adolescents / Body mass index / Cardiometabolic risk factors / Children / Classification / Cut-offs / Obesity / Overweight
  • [Investigator] Comici A; Corciulo N; Di Bonito P; Di Candia S; Franzese A; Modestini E; Morino G; Moro B; Tanas R
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5. Smith C: Prevalence of obesity and contributing factors among Sherpa women in urban and rural Nepal. Am J Hum Biol; 1998;10(4):519-528

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Prevalence of obesity and contributing factors among Sherpa women in urban and rural Nepal.
  • This report used data from a prospective longitudinal study to assess the prevalence of obesity among Sherpa women and to determine which factors were most predictive of it.
  • Although mean BMI was in the range associated with the overall lowest risk to health, distribution of the BMI showed that 29% of the urban low altitude sample were classified obese.
  • The prevalence of obesity was correlated with reduced energy expenditure, not to an increase in consumed calories.
  • These data suggest that Sherpa women may potentially be at risk of chronic health problems in the future because of increased adiposity. Am. J. Hum. Biol.

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  • [Copyright] Copyright © 1998 Wiley-Liss, Inc.
  • (PMID = 28561471.001).
  • [ISSN] 1520-6300
  • [Journal-full-title] American journal of human biology : the official journal of the Human Biology Council
  • [ISO-abbreviation] Am. J. Hum. Biol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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6. Chang YH: Common therapeutic target for both cancer and obesity. World J Biol Chem; 2017 May 26;8(2):102-107

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Common therapeutic target for both cancer and obesity.
  • Obesity and cancer are two interrelated conditions of high epidemiological need, with studies showing that obesity is responsible for nearly 25% of the relative contribution to cancer incidence.
  • Given the connection between these conditions, a drug that can operate on both obesity and cancer is highly desirable.
  • Though this lack of success could be attributed to off-target adverse effects, the underlying causes remain unclear.
  • However, due to insufficient knowledge of the biological functions of N-terminal protein processing, it is hard to predict whether these novel inhibitors would successfully pass clinical trials and thereby benefit cancer and obesity patients.

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  • [Cites] J Bacteriol. 1987 Feb;169(2):751-7 [3027045.001]
  • [Cites] Proc Natl Acad Sci U S A. 1997 Jun 10;94(12 ):6099-103 [9177176.001]
  • [Cites] J Biol Chem. 1990 Nov 15;265(32):19892-7 [2246265.001]
  • [Cites] Front Pharmacol. 2017 Jan 06;7:519 [28111549.001]
  • [Cites] Cancer Res. 1994 Apr 15;54(8):2073-6 [7513609.001]
  • [Cites] Biochem Biophys Res Commun. 1991 Feb 14;174(3):1070-6 [1705118.001]
  • [Cites] Invest New Drugs. 2012 Apr;30(2):672-80 [21188464.001]
  • [Cites] J Biol Chem. 2005 Apr 8;280(14):14356-60 [15699032.001]
  • [Cites] Arch Biochem Biophys. 2002 Feb 1;398(1):87-93 [11811952.001]
  • [Cites] Cell. 1996 Aug 9;86(3):353-64 [8756718.001]
  • [Cites] Nat Med. 1995 Jan;1(1):27-31 [7584949.001]
  • [Cites] Chem Rev. 2002 Dec;102(12):4581-608 [12475202.001]
  • [Cites] Biochimie. 2000 Feb;82(2):95-107 [10727764.001]
  • [Cites] J Clin Invest. 2003 Mar;111(5):565-70 [12618507.001]
  • [Cites] Proc Natl Acad Sci U S A. 1995 Aug 15;92(17):7714-8 [7644482.001]
  • [Cites] Int J Oncol. 2006 Apr;28(4):955-63 [16525646.001]
  • [Cites] Biochemistry. 1987 Dec 15;26(25):8242-6 [3327521.001]
  • [Cites] Nature. 1990 Dec 6;348(6301):555-7 [1701033.001]
  • [Cites] Curr Med Chem. 2012;19(7):1021-35 [22229417.001]
  • [Cites] Nature. 1997 Apr 17;386(6626):671-4 [9109485.001]
  • [Cites] N Engl J Med. 1971 Nov 18;285(21):1182-6 [4938153.001]
  • [Cites] Bioorg Med Chem. 1998 Aug;6(8):1163-9 [9784858.001]
  • [Cites] Proc Natl Acad Sci U S A. 2000 Jun 6;97(12 ):6427-32 [10841547.001]
  • [Cites] Chem Biol. 1999 Nov;6(11):823-33 [10574784.001]
  • [Cites] Curr Pharm Des. 2016;22(7):895-903 [26648466.001]
  • [Cites] Arthritis Rheum. 2007 Mar;56(3):850-60 [17328059.001]
  • [Cites] J Clin Invest. 2007 Sep;117(9):2362-8 [17786229.001]
  • [Cites] Bioorg Med Chem Lett. 2016 Jun 15;26(12):2779-83 [27136719.001]
  • [Cites] J Clin Oncol. 1999 Aug;17 (8):2541-5 [10561320.001]
  • [Cites] J Biol Chem. 1990 Nov 15;265(32):19638-43 [2174047.001]
  • [Cites] Bioorg Med Chem Lett. 2016 Jun 15;26(12):2774-8 [27155900.001]
  • [Cites] EMBO J. 2003 Jan 2;22(1):13-23 [12505980.001]
  • [Cites] J Biol Chem. 1985 May 10;260(9):5382-91 [2985590.001]
  • [Cites] Nat Rev Drug Discov. 2010 Feb;9(2):107-15 [20118961.001]
  • [Cites] Mol Gen Genet. 1995 Jan 20;246(2):247-53 [7862096.001]
  • [Cites] Clin Cancer Res. 2006 Apr 15;12(8):2583-90 [16638869.001]
  • [Cites] Nature. 2000 Apr 6;404(6778):635-43 [10766250.001]
  • [Cites] J Exp Med. 1972 Aug 1;136(2):261-76 [5043412.001]
  • [Cites] J Cell Biochem. 2005 Mar 1;94(4):656-68 [15547949.001]
  • [Cites] J Bacteriol. 1989 Jul;171(7):4071-2 [2544569.001]
  • [Cites] ChemMedChem. 2013 May;8(5):819-31 [23512621.001]
  • [Cites] Adv Cancer Res. 1985;43:175-203 [2581424.001]
  • [Cites] Biochem Biophys Res Commun. 2013 Oct 11;440(1):37-42 [24041691.001]
  • [Cites] Proc Natl Acad Sci U S A. 2008 Feb 12;105(6):1838-43 [18252827.001]
  • [Cites] J Med Chem. 2007 Aug 9;50(16):3777-85 [17636946.001]
  • [Cites] Diabetes Metab Syndr Obes. 2014 Feb 28;7:73-84 [24611021.001]
  • [Cites] Chem Pharm Bull (Tokyo). 1992 Jan;40(1):96-101 [1374294.001]
  • [Cites] J Biol Chem. 1992 Apr 25;267(12):8007-11 [1569059.001]
  • [Cites] Nature. 2000 Apr 6;404(6778):632-4 [10766249.001]
  • [Cites] J Cell Biochem. 2002;85(4):678-88 [11968008.001]
  • [Cites] N Engl J Med. 1995 Dec 28;333(26):1757-63 [7491141.001]
  • [Cites] Chem Biol. 1997 Jun;4(6):461-71 [9224570.001]
  • [Cites] J Cell Biol. 2015 Jul 20;210(2):287-301 [26195668.001]
  • [Cites] Biochimie. 2015 Jul;114:134-46 [25450248.001]
  • [Cites] Expert Rev Anti Infect Ther. 2007 Aug;5(4):573-9 [17678422.001]
  • [Cites] Proc Natl Acad Sci U S A. 1989 Nov;86(21):8247-51 [2682640.001]
  • [Cites] Proc Natl Acad Sci U S A. 1987 May;84(9):2718-22 [3106976.001]
  • [Cites] J Biol Chem. 1992 Oct 15;267(29):20667-73 [1328207.001]
  • [Cites] Arthritis Res Ther. 2007;9(6):R127 [18072970.001]
  • [Cites] Arthritis Rheum. 2010 Jul;62(7):1890-8 [20309868.001]
  • [Cites] Proc Natl Acad Sci U S A. 1995 Dec 19;92(26):12357-61 [8618900.001]
  • [Cites] Biochim Biophys Acta. 1995 Feb 21;1260(3):333-6 [7873610.001]
  • [Cites] Oncotarget. 2016 Sep 27;7(39):63306-63323 [27542228.001]
  • [Cites] J Med Chem. 2009 Dec 24;52(24):8047-56 [19929003.001]
  • [Cites] Nat Commun. 2014 Jun 18;5:4180 [24939037.001]
  • [Cites] Bioorg Med Chem Lett. 2006 May 15;16(10):2580-3 [16540317.001]
  • [Cites] Cancer Cell. 2005 Mar;7(3):251-61 [15766663.001]
  • [Cites] Science. 1998 Nov 13;282(5392):1324-7 [9812898.001]
  • [Cites] Antibiot Chemother (Northfield). 1951 Apr;1(1):54-8 [24540610.001]
  • [Cites] Anal Biochem. 1994 Nov 1;222(2):514-6 [7864384.001]
  • [Cites] Proc Natl Acad Sci U S A. 2004 Jul 20;101(29):10768-73 [15249666.001]
  • (PMID = 28588753.001).
  • [ISSN] 1949-8454
  • [Journal-full-title] World journal of biological chemistry
  • [ISO-abbreviation] World J Biol Chem
  • [Language] eng
  • [Publication-type] Editorial
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Angiogenesis / Cancer / Diabetes / Methionine aminopeptidase / Obesity / Protein maturation / Protein modification / Protein processing / Protein stability
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7. Zhu A, Chen J, Wu P, Luo M, Zeng Y, Liu Y, Zheng H, Zhang L, Chen Z, Sun Q, Li W, Duan Y, Su D, Xiao Z, Duan Z, Zheng S, Bai L, Zhang X, Ju Z, Li Y, Hu R, Pandol SJ, Han YP: Cationic Polystyrene Resolves Nonalcoholic Steatohepatitis, Obesity, and Metabolic Disorders by Promoting Eubiosis of Gut Microbiota and Decreasing Endotoxemia. Diabetes; 2017 Aug;66(8):2137-2143
MedlinePlus Health Information. consumer health - Obesity.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Cationic Polystyrene Resolves Nonalcoholic Steatohepatitis, Obesity, and Metabolic Disorders by Promoting Eubiosis of Gut Microbiota and Decreasing Endotoxemia.
  • A pandemic of metabolic diseases, consisting of type 2 diabetes, nonalcoholic fatty liver disease, and obesity, has imposed critical challenges for societies worldwide, prompting investigation of underlying mechanisms and exploration of low-cost and effective treatment.
  • [MeSH-major] Cation Exchange Resins / administration & dosage. Endotoxemia / drug therapy. Gastrointestinal Microbiome / drug effects. Metabolic Diseases / therapy. Non-alcoholic Fatty Liver Disease / drug therapy. Obesity / drug therapy. Polystyrenes / administration & dosage
  • [MeSH-minor] Administration, Oral. Animals. Diet, High-Fat / adverse effects. Dysbiosis / drug therapy. Dysbiosis / microbiology. Endotoxins / blood. Fecal Microbiota Transplantation / methods. Insulin Resistance. Male. Mice, Inbred C57BL. Symbiosis / drug effects. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Fatty Liver Disease.
  • MedlinePlus Health Information. consumer health - Metabolic Disorders.
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  • [Copyright] © 2017 by the American Diabetes Association.
  • [Cites] J Gastroenterol Hepatol. 2014 Jun;29(6):1292-8 [24372861.001]
  • [Cites] J Gastroenterol Hepatol. 2013 Aug;28 Suppl 1:49-55 [23855296.001]
  • [Cites] Aliment Pharmacol Ther. 2015 Sep;42(6):731-40 [26202818.001]
  • [Cites] Mayo Clin Proc. 2006 Mar;81(3):353-73 [16529140.001]
  • [Cites] Nature. 2013 Aug 29;500(7464):541-6 [23985870.001]
  • [Cites] Nature. 2006 Dec 21;444(7122):1027-31 [17183312.001]
  • [Cites] Clin Liver Dis. 2012 Aug;16(3):549-65 [22824480.001]
  • [Cites] PLoS One. 2012;7(8):e38286 [22952571.001]
  • [Cites] BMC Med. 2011 Jul 12;9:85 [21749681.001]
  • [Cites] Nutr Metab (Lond). 2010 Sep 03;7:73 [20815878.001]
  • [Cites] Front Physiol. 2016 Nov 15;7:498 [27895587.001]
  • [Cites] J Diabetes. 2010 Sep;2(3):180-93 [20923483.001]
  • [Cites] Hepatology. 2010 Nov;52(5):1836-46 [21038418.001]
  • [Cites] J Clin Invest. 2006 Jul;116(7):1793-801 [16823477.001]
  • [Cites] Endocrinol Metab Clin North Am. 2004 Jun;33(2):351-75, table of contents [15158523.001]
  • [Cites] Gastroenterology. 1998 Apr;114(4):842-5 [9547102.001]
  • [Cites] Hepatol Res. 2009 Jul;39(7):685-93 [19473432.001]
  • [Cites] Diabetes. 2007 Jul;56(7):1761-72 [17456850.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2014 Nov 1;307(9):G883-93 [25214402.001]
  • [Cites] Front Physiol. 2013 Jan 31;3:496 [23386831.001]
  • [Cites] Nature. 2009 Jan 22;457(7228):480-4 [19043404.001]
  • (PMID = 28446519.001).
  • [ISSN] 1939-327X
  • [Journal-full-title] Diabetes
  • [ISO-abbreviation] Diabetes
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / P01 CA163200; United States / NIDDK NIH HHS / DK / P01 DK098108
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cation Exchange Resins; 0 / Endotoxins; 0 / Polystyrenes
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8. Announcement: Community Preventive Services Task Force Recommendation for Interventions Including Activity Monitors to Increase Physical Activity in Adults with Overweight or Obesity. MMWR Morb Mortal Wkly Rep; 2017 Nov 03;66(43):1203

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Announcement: Community Preventive Services Task Force Recommendation for Interventions Including Activity Monitors to Increase Physical Activity in Adults with Overweight or Obesity.

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  • (PMID = 29095801.001).
  • [ISSN] 1545-861X
  • [Journal-full-title] MMWR. Morbidity and mortality weekly report
  • [ISO-abbreviation] MMWR Morb. Mortal. Wkly. Rep.
  • [Language] eng
  • [Publication-type] News
  • [Publication-country] United States
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9. Farrell P, Thow AM, Abimbola S, Faruqui N, Negin J: How food insecurity could lead to obesity in LMICs:  When not enough is too much: a realist review of how food insecurity could lead to obesity in low- and middle-income countries. Health Promot Int; 2017 May 24;

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] How food insecurity could lead to obesity in LMICs:  When not enough is too much: a realist review of how food insecurity could lead to obesity in low- and middle-income countries.
  • While food insecurity is a well-known cause of under-nutrition and stunting, in recent decades it has also been linked with obesity.
  • Understanding and thus minimising the risk factors for obesity in low- and middle-income country contexts, which often lack the health system capacity to treat the consequent obesity-related illnesses, is crucial.
  • By comparing this evidence to existing theory on food security and nutrition, we aimed to provide a richer understanding of the nuances and the socio-demographic nature of the food insecurity-obesity relationship.
  • The review generated 13 peer-reviewed articles which studied the relationship between food insecurity and overweight/obesity in low- and middle-income countries.
  • Affordability of high-energy, processed foods was identified as a main mechanism, which determined whether or not food insecurity leads to obesity in low- and middle-income countries.
  • Other mechanisms identified were: quantity and diversity of food consumed; spatial-temporal access to nutritious food; inter-personal food choice and distribution; and non-dietary behaviours.
  • Despite the limited empirical evidence available, our review presents meaningful and policy-relevant insights into the food insecurity-obesity relationship in from low- and middle-income countries.
  • Interventions to address the food insecurity-obesity link need to address diet quality, and demand a broad understanding across a variety of experiences.

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  • [Copyright] © The Author 2017. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com.
  • (PMID = 28541498.001).
  • [ISSN] 1460-2245
  • [Journal-full-title] Health promotion international
  • [ISO-abbreviation] Health Promot Int
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Keywords] NOTNLM ; food insecurity / low-income country / middle-income country / obesity / overweight
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10. Velazquez-Bautista M, López-Sandoval JJ, González-Hita M, Vázquez-Valls E, Cabrera-Valencia IZ, Torres-Mendoza BM: Association of metabolic syndrome with low birth weight, intake of high-calorie diets and acanthosis nigricans in children and adolescents with overweight and obesity. Endocrinol Diabetes Nutr; 2017 Jan;64(1):11-17

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Association of metabolic syndrome with low birth weight, intake of high-calorie diets and acanthosis nigricans in children and adolescents with overweight and obesity.
  • [Transliterated title] Asociación del síndrome metabólico con bajo peso al nacimiento, consumo de dietas hipercalóricas y acantosis nigricans en escolares y adolescentes con sobrepeso y obesidad.
  • OBJECTIVE: The theory of fetal programming suggests that low birth weight (LBW) predisposes to greater food intake and increases the chance of overweight and obesity, which are in turn associated to conditions such as metabolic syndrome (MS) and acanthosis nigricans.
  • The study objective was to ascertain whether an association exists between MS, LBW, intake of high-calorie diets, and acanthosis nigricans in children and adolescents with overweight or obesity.
  • MATERIAL AND METHODS: A case-control was conducted on 100 children who attended the overweight and obesity outpatient clinic of the OPD Hospital Civil de Guadalajara "Fray Antonio Alcalde".
  • RESULTS: The results show that 82% of children and adolescents were obese and 18% overweight, and 73% had MS.
  • CONCLUSIONS: In children and adolescents with overweight and obesity, LBW, high-calorie diets, and acanthosis nigricans are associated to a higher probability of MS.

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  • [Copyright] Copyright © 2016 SEEN. Publicado por Elsevier España, S.L.U. All rights reserved.
  • (PMID = 28440765.001).
  • [ISSN] 2530-0164
  • [Journal-full-title] Endocrinologia, diabetes y nutricion
  • [ISO-abbreviation] Endocrinol Diabetes Nutr
  • [Language] eng; spa
  • [Publication-type] Journal Article
  • [Publication-country] Spain
  • [Keywords] NOTNLM ; Acanthosis nigricans / Acantosis nigricans / Bajo peso al nacimiento / Dieta hipercalórica / High-calorie diet / Low birth weight / Metabolic syndrome / Obesidad / Obesity / Síndrome metabólico
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