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Items 1 to 10 of about 4835
1. Li X, Xu K, Zhang Y, Sun C, He Y: Optical Determination of Lead Chrome Green in Green Tea by Fourier Transform Infrared (FT-IR) Transmission Spectroscopy. PLoS One; 2017;12(1):e0169430

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Optical Determination of Lead Chrome Green in Green Tea by Fourier Transform Infrared (FT-IR) Transmission Spectroscopy.
  • The potential of Fourier transform infrared (FT-IR) transmission spectroscopy for determination of lead chrome green in green tea was investigated based on chemometric methods.
  • Firstly, the qualitative analysis of lead chrome green in tea was performed based on partial least squares discriminant analysis (PLS-DA), and the correct rate of classification was 100%.
  • And then, a hybrid method of interval partial least squares (iPLS) regression and successive projections algorithm (SPA) was proposed to select characteristic wavenumbers for the quantitative analysis of lead chrome green in green tea, and 19 wavenumbers were obtained finally.
  • Among these wavenumbers, 1384 (C = C), 1456, 1438, 1419(C = N), and 1506 (CNH) cm-1 were the characteristic wavenumbers of lead chrome green.
  • All these results indicated the feasibility of IR spectra for detecting lead chrome green in green tea.

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  • [Cites] Food Chem. 2015 Sep 15;183:30-5 [25863606.001]
  • [Cites] Spectrochim Acta A Mol Biomol Spectrosc. 2013 Jul;111:31-6 [23602956.001]
  • [Cites] Food Chem. 2015 Jul 15;179:175-81 [25722152.001]
  • [Cites] Food Chem. 2014 Sep 1;158:351-7 [24731354.001]
  • [Cites] Anal Chim Acta. 2008 May 12;615(1):10-7 [18440358.001]
  • [Cites] Sci Rep. 2015 Oct 28;5:15729 [26508516.001]
  • [Cites] Anal Chim Acta. 2009 Apr 6;638(1):16-22 [19298874.001]
  • [Cites] Food Chem. 2015 Jun 1;176:130-6 [25624215.001]
  • [Cites] Neural Netw. 2001 Jan;14(1):23-35 [11213211.001]
  • [Cites] Biol Trace Elem Res. 2010 Aug;136(2):127-39 [20195925.001]
  • [Cites] Food Chem Toxicol. 2014 Mar;65:227-32 [24394486.001]
  • [Cites] Appl Spectrosc. 2007 Mar;61(3):293-9 [17389069.001]
  • [Cites] Planta Med. 2014 Aug;80(12):1023-8 [25098931.001]
  • (PMID = 28068348.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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2. Farhan M, Oves M, Chibber S, Hadi SM, Ahmad A: Mobilization of Nuclear Copper by Green Tea Polyphenol Epicatechin-3-Gallate and Subsequent Prooxidant Breakage of Cellular DNA: Implications for Cancer Chemotherapy. Int J Mol Sci; 2016 Dec 26;18(1)

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mobilization of Nuclear Copper by Green Tea Polyphenol Epicatechin-3-Gallate and Subsequent Prooxidant Breakage of Cellular DNA: Implications for Cancer Chemotherapy.
  • Epidemiological as well as experimental evidence exists in support of chemopreventive and anticancer properties of green tea and its constituents.
  • The gallocatechin, epicatechin-3-gallate is a major polyphenol present in green tea, shown responsible for these effects.
  • Our results are indicative of ROS generation, possibly through mobilization of endogenous copper ions, and support our long-standing hypothesis of a prooxidant activity of plant-derived polyphenols as a mechanism for their documented anticancer properties.
  • [MeSH-minor] Cell Line, Tumor. Cells, Cultured. Humans. Lymphocytes / drug effects. Lymphocytes / metabolism. Reactive Oxygen Species / metabolism. Tea / chemistry

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  • [Cites] Cancer Lett. 2000 Jun 1;154(1):29-37 [10799736.001]
  • [Cites] Biochem Biophys Res Commun. 1984 Aug 30;123(1):291-8 [6477583.001]
  • [Cites] Annu Rev Biochem. 1980;49:695-726 [6250449.001]
  • [Cites] Free Radic Biol Med. 1994 Jan;16(1):43-8 [8299995.001]
  • [Cites] Food Chem Toxicol. 1993 Apr;31(4):271-83 [7682977.001]
  • [Cites] Biochem J. 1997 Apr 15;323 ( Pt 2):337-41 [9163321.001]
  • [Cites] J Biol Chem. 2010 Nov 5;285(45):34557-65 [20826787.001]
  • [Cites] J Natl Cancer Inst. 1997 Dec 17;89(24):1881-6 [9414176.001]
  • [Cites] Stem Cells. 1995 May;13 Suppl 1:207-14 [7488947.001]
  • [Cites] Clin Cancer Res. 2011 Aug 15;17(16):5402-11 [21705453.001]
  • [Cites] Blood. 1998 Aug 1;92(3):996-1002 [9680369.001]
  • [Cites] Mutat Res. 1994 Aug;313(1):39-48 [7519309.001]
  • [Cites] FEBS Lett. 2006 Jan 23;580(2):533-8 [16412432.001]
  • [Cites] Cancer Lett. 1998 Jul 17;129(2):173-9 [9719459.001]
  • [Cites] Open Biol. 2013 Jan 08;3(1):120144 [23303309.001]
  • [Cites] J Oncol. 2012;2012:749310 [23316231.001]
  • [Cites] Biochim Biophys Acta. 1996 Nov 15;1317(2):95-100 [8950193.001]
  • [Cites] Mutagenesis. 1998 May;13(3):271-4 [9643586.001]
  • [Cites] J Biol Chem. 1991 Oct 25;266(30):20175-84 [1939078.001]
  • [Cites] Toxicology. 2006 Aug 15;225(2-3):173-82 [16843582.001]
  • [Cites] Free Radic Biol Med. 2002 Aug 1;33(3):387-98 [12126761.001]
  • [Cites] Mol Nutr Food Res. 2011 Apr;55(4):553-9 [21462322.001]
  • [Cites] Cancer Cell. 2006 Sep;10(3):241-52 [16959615.001]
  • [Cites] Free Radic Biol Med. 1999 Sep;27(5-6):612-6 [10490282.001]
  • [Cites] FEBS Lett. 2005 Jun 6;579(14 ):3131-5 [15919081.001]
  • [Cites] Biochem Biophys Res Commun. 2000 Jun 24;273(1):50-3 [10873562.001]
  • [Cites] IUBMB Life. 2000 Sep;50(3):167-71 [11142343.001]
  • [Cites] Free Radic Biol Med. 1994 Jun;16(6):845-50 [8070690.001]
  • [Cites] Mol Nutr Food Res. 2014 Mar;58(3):437-46 [24123728.001]
  • [Cites] Nature. 2012 Apr 18;486(7403):346-52 [22522925.001]
  • [Cites] Cancer Res. 1988 May 1;48(9):2361-5 [3128399.001]
  • [Cites] Biochem Biophys Res Commun. 1994 Oct 28;204(2):898-904 [7980558.001]
  • [Cites] Carcinogenesis. 1992 Apr;13(4):605-8 [1315626.001]
  • [Cites] Biochem Pharmacol. 1987 Nov 1;36(21):3629-33 [2823829.001]
  • [Cites] Cancer Lett. 1998 Feb 13;124(1):23-30 [9500187.001]
  • [Cites] Food Chem Toxicol. 1992 Jun;30(6):483-9 [1379971.001]
  • [Cites] Free Radic Res. 2008 Aug;42(8):764-72 [18661438.001]
  • [Cites] Phytother Res. 2003 Apr;17(4):358-63 [12722140.001]
  • [Cites] Cancer Cell. 2006 Sep;10(3):175-6 [16959608.001]
  • (PMID = 28035959.001).
  • [ISSN] 1422-0067
  • [Journal-full-title] International journal of molecular sciences
  • [ISO-abbreviation] Int J Mol Sci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Antineoplastic Agents, Phytogenic; 0 / Reactive Oxygen Species; 0 / Tea; 789U1901C5 / Copper; 8R1V1STN48 / Catechin; 92587OVD8Z / epicatechin gallate
  • [Keywords] NOTNLM ; DNA damage (major topic) / anticancer (major topic) / comet assay (major topic) / copper (major topic) / epicatechin-3-gallate (major topic) / prooxidant (major topic) / reactive oxygen species (major topic)
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3. Matsuo T, Miyata Y, Asai A, Sagara Y, Furusato B, Fukuoka J, Sakai H: Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer. PLoS One; 2017;12(1):e0171091
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer.
  • Green tea polyphenol (GTP) suppresses carcinogenesis and aggressiveness in many types of malignancies including bladder cancer.

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  • [Cites] Life Sci. 2013 Sep 3;93(8):307-12 [23871988.001]
  • [Cites] DNA Cell Biol. 2003 Mar;22(3):217-24 [12804120.001]
  • [Cites] J Biol Chem. 2006 Nov 3;281(44):33761-72 [16950787.001]
  • [Cites] PLoS One. 2013;8(3):e59095 [23516604.001]
  • [Cites] Cancer Res. 2006 Jan 15;66(2):1234-40 [16424063.001]
  • [Cites] Vasc Cell. 2013 May 02;5(1):9 [23638734.001]
  • [Cites] J Nutr. 2002 Aug;132(8):2307-11 [12163680.001]
  • [Cites] Urol Int. 2013;90(1):10-6 [23052791.001]
  • [Cites] Oncotarget. 2015 Sep 29;6(29):27312-31 [26314962.001]
  • [Cites] Cancer Epidemiol. 2010 Jun;34(3):350-4 [20362526.001]
  • [Cites] Carcinogenesis. 2004 Nov;25(11):2217-24 [15358631.001]
  • [Cites] Biochem Pharmacol. 2011 Dec 15;82(12):1807-21 [21827739.001]
  • [Cites] Br J Cancer. 2001 Mar 23;84(6):844-50 [11259102.001]
  • [Cites] Clin Cancer Res. 2003 May;9(5):1741-9 [12738729.001]
  • [Cites] Clin Cancer Res. 2007 Dec 1;13(23):6959-63 [18056170.001]
  • [Cites] Urology. 2011 Aug;78(2):476.e9-15 [21696810.001]
  • [Cites] Cancer Lett. 2001 Jun 26;167(2):175-82 [11369139.001]
  • [Cites] Mol Cell Biol. 2009 May;29(10 ):2622-35 [19289500.001]
  • [Cites] Cancer Res Treat. 2009 Jun;41(2):87-92 [19707506.001]
  • [Cites] Cancer Epidemiol. 2015 Dec;39 Suppl 1:S84-92 [26164655.001]
  • [Cites] Rev Urol. 2006 Winter;8(1):8-13 [16985555.001]
  • [Cites] Biochem Biophys Res Commun. 2007 Mar 23;354(4):852-7 [17266926.001]
  • [Cites] Sci Rep. 2015 Mar 12;5:9051 [25761588.001]
  • [Cites] Tumour Biol. 2015 Jan;36(1):315-27 [25252849.001]
  • [Cites] Biomed Res. 2009 Aug;30(4):207-15 [19729851.001]
  • [Cites] Basic Clin Pharmacol Toxicol. 2015 Sep;117(3):164-72 [25625183.001]
  • [Cites] Transl Res. 2014 Dec;164(6):468-76 [25063314.001]
  • [Cites] Clin Nutr. 2013 Dec;32(6):894-903 [23582951.001]
  • [Cites] Anticancer Res. 2014 Feb;34(2):735-44 [24511007.001]
  • [Cites] J Natl Cancer Inst. 2007 Apr 4;99(7):558-68 [17406000.001]
  • [Cites] Life Sci. 2006 Nov 17;79(25):2329-36 [16945390.001]
  • [Cites] J Neurooncol. 2008 Jun;88(2):143-55 [18317686.001]
  • [Cites] Biochem Biophys Res Commun. 2012 Nov 2;427(4):725-30 [23036202.001]
  • [Cites] Med Oncol. 2011 Dec;28 Suppl 1:S577-85 [21046284.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2015 Oct;24(10):1516-22 [26215293.001]
  • [Cites] Am J Transl Res. 2016 Feb 15;8(2):578-87 [27158349.001]
  • [Cites] Clin Cancer Res. 2006 Feb 1;12(3 Pt 1):800-6 [16467091.001]
  • [Cites] Tumour Biol. 2016 Apr;37(4):4373-82 [26499783.001]
  • [Cites] Mol Cancer Res. 2011 May;9(5):648-59 [21498545.001]
  • [Cites] PLoS One. 2011;6(10):e25224 [22022384.001]
  • [Cites] Nutr Cancer. 2014;66(2):315-24 [24447094.001]
  • [Cites] J Gastroenterol. 2014 Apr;49(4):692-701 [23543313.001]
  • [Cites] Oncol Rep. 2015 Jun;33(6):2972-80 [25845434.001]
  • [Cites] Int J Cancer. 1997 Jan 27;70(3):255-8 [9033623.001]
  • [Cites] Leuk Res. 2007 Sep;31(9):1277-84 [17081606.001]
  • (PMID = 28141864.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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4. Maiti S, Acharyya N, Ghosh TK, Ali SS, Manna E, Nazmeen A, Sinha NK: Green Tea (Camellia sinensis) Protects Against Arsenic Neurotoxicity via Antioxidative Mechanism And Activation of Superoxide Dismutase Activity. Cent Nerv Syst Agents Med Chem; 2017 Feb 01;

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea (Camellia sinensis) Protects Against Arsenic Neurotoxicity via Antioxidative Mechanism And Activation of Superoxide Dismutase Activity.
  • Here, the protective role of Green tea (Camellia sinensis or CS; 10mg/ml aqueous) has been evaluated against arsenic-induced (0.6ppm/100g bw/28 days) cerebral/cerebellar tissue degeneration, oxidative-threats and neurotransmitter deregulation in female rats.

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  • (PMID = 28155600.001).
  • [ISSN] 1875-6166
  • [Journal-full-title] Central nervous system agents in medicinal chemistry
  • [ISO-abbreviation] Cent Nerv Syst Agents Med Chem
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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5. Li J, Sapper TN, Mah E, Moller MV, Kim JB, Chitchumroonchokchai C, McDonald JD, Bruno RS: Green tea extract treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by lowering TNFR1 and TLR4 expression and ligand availability. J Nutr Biochem; 2017 Mar;41:34-41

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea extract treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by lowering TNFR1 and TLR4 expression and ligand availability.
  • We hypothesized that antiinflammatory activities of green tea extract (GTE) during NASH would lower tumor necrosis factor receptor-1 (TNFR1)- and Toll-like receptor-4 (TLR4)-mediated NFκB activation.
  • These data suggest that dietary GTE treatment reduces hepatic inflammation in NASH by decreasing proinflammatory signaling through TNFR1 and TLR4 that otherwise increases NFκB activation and liver injury.

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  • [Copyright] Copyright © 2016 Elsevier Inc. All rights reserved.
  • (PMID = 28038359.001).
  • [ISSN] 1873-4847
  • [Journal-full-title] The Journal of nutritional biochemistry
  • [ISO-abbreviation] J. Nutr. Biochem.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Green tea / Inflammation / NASH / Nonalcoholic steatohepatitis / TLR4 / TNFR1
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6. Bao H, Peng A: The Green Tea Polyphenol(-)-epigallocatechin-3-gallate and its beneficial roles in chronic kidney disease. J Transl Int Med; 2016 Sep 01;4(3):99-103
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The Green Tea Polyphenol(-)-epigallocatechin-3-gallate and its beneficial roles in chronic kidney disease.
  • In particular, oxidative stress as well as inflammation appears to play a pivotal role in CKD progression. ()-Epigallocatechin-3-gallate (EGCG), the major catechin of green tea extract, is known as a powerful antioxidant and reactive oxygen species scavenger.

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  • (PMID = 28191529.001).
  • [ISSN] 2450-131X
  • [Journal-full-title] Journal of translational internal medicine
  • [ISO-abbreviation] J Transl Int Med
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Germany
  • [Keywords] NOTNLM ; EGCG / apoptosis / chronic kidney disease / green tea / inflammation
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7. Kumazoe M, Nakamura Y, Yamashita M, Suzuki T, Takamatsu K, Huang Y, Bae J, Yamashita S, Murata M, Yamada S, Shinoda Y, Yamaguchi W, Toyoda Y, Tachibana H: Green Tea Polyphenol Epigallocatechin-3-gallate Suppresses Toll-like Receptor 4 Expression via Up-regulation of E3 Ubiquitin-protein Ligase RNF216. J Biol Chem; 2017 Mar 10;292(10):4077-4088
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea Polyphenol Epigallocatechin-3-gallate Suppresses Toll-like Receptor 4 Expression via Up-regulation of E3 Ubiquitin-protein Ligase RNF216.

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  • [Copyright] © 2017 by The American Society for Biochemistry and Molecular Biology, Inc.
  • [Cites] Curr Diab Rep. 2009 Feb;9(1):26-32 [19192421.001]
  • [Cites] Diabetol Metab Syndr. 2012 Jul 04;4(1):32 [22762794.001]
  • [Cites] Proc Natl Acad Sci U S A. 2015 Mar 31;112(13):4086-91 [25775533.001]
  • [Cites] Cytotechnology. 2007 Dec;55(2-3):135-42 [19003003.001]
  • [Cites] Curr Opin Lipidol. 2009 Feb;20(1):39-44 [19133410.001]
  • [Cites] Crit Care. 2011;15(2):R109 [21466684.001]
  • [Cites] Nat Med. 2012 Aug;18(8):1279-85 [22842477.001]
  • [Cites] Lancet. 2005 Jan 1-7;365(9453):36-42 [15639678.001]
  • [Cites] Biochem J. 2012 Apr 15;443(2):525-34 [22257159.001]
  • [Cites] J Clin Invest. 2006 Nov;116(11):3015-25 [17053832.001]
  • [Cites] Mol Med Rep. 2014 Sep;10(3):1383-8 [25059833.001]
  • [Cites] PLoS One. 2007 Nov 07;2(11):e1153 [17987129.001]
  • [Cites] Circ Res. 2007 Jun 8;100(11):1589-96 [17478729.001]
  • [Cites] FEBS Lett. 2013 Sep 17;587(18):3052-7 [23916810.001]
  • [Cites] Trends Endocrinol Metab. 2011 Jan;22(1):16-23 [20888253.001]
  • [Cites] Diabetes. 2000 Nov;49(11):1880-9 [11078455.001]
  • [Cites] Biochem Biophys Res Commun. 2012 Oct 5;426(4):480-5 [22960171.001]
  • [Cites] Proc Natl Acad Sci U S A. 2004 Jul 20;101(29):10679-84 [15249654.001]
  • [Cites] FEBS J. 2009 Feb;276(3):616-21 [19143830.001]
  • [Cites] Am J Physiol Cell Physiol. 2000 Oct;279(4):C1100-6 [11003590.001]
  • [Cites] Diabetes. 1992 Oct;41 Suppl 2:97-101 [1526345.001]
  • [Cites] J Clin Oncol. 2009 Aug 10;27(23):3808-14 [19470922.001]
  • [Cites] Int J Vasc Med. 2012;2012:918267 [22611498.001]
  • [Cites] Circulation. 2009 Oct 20;120(16):1640-5 [19805654.001]
  • [Cites] Br J Pharmacol. 2012 Mar;165(5):1288-305 [22014080.001]
  • [Cites] J Clin Invest. 2006 Jun;116(6):1494-505 [16691291.001]
  • [Cites] J Neuroinflammation. 2012 Jul 06;9:161 [22768975.001]
  • [Cites] J Clin Invest. 2005 Apr;115(4):1030-8 [15761499.001]
  • [Cites] J Clin Invest. 2003 Dec;112(12):1796-808 [14679176.001]
  • [Cites] Sci Rep. 2013 Sep 25;3:2749 [24067358.001]
  • [Cites] Metabolism. 1998 Jan;47(1):113-8 [9440488.001]
  • [Cites] Obesity (Silver Spring). 2011 Jun;19(6):1109-17 [21331068.001]
  • [Cites] Obesity (Silver Spring). 2013 Jun;21(6):1208-14 [23913732.001]
  • [Cites] Asian J Androl. 2008 Nov;10(6):905-13 [18097500.001]
  • [Cites] Diabetes. 2003 May;52(5):1280-3 [12716765.001]
  • [Cites] Eur Cytokine Netw. 2006 Mar;17(1):4-12 [16613757.001]
  • [Cites] Metabolism. 2002 Sep;51(9):1104-10 [12200753.001]
  • [Cites] J Clin Invest. 2013 Feb;123(2):787-99 [23348740.001]
  • [Cites] J Neuroinflammation. 2011 Aug 09;8:92 [21827663.001]
  • [Cites] Anticancer Res. 2004 Mar-Apr;24(2B):747-53 [15161022.001]
  • [Cites] Nat Genet. 2001 Jun;28(2):178-83 [11381268.001]
  • [Cites] J Am Coll Nutr. 2008 Feb;27(1):1-5 [18460475.001]
  • [Cites] Proc Natl Acad Sci U S A. 2012 Apr 17;109(16):6325-30 [22474354.001]
  • [Cites] Nat Struct Mol Biol. 2004 Apr;11(4):380-1 [15024383.001]
  • [Cites] Sci Rep. 2015 Mar 31;5:9474 [25824377.001]
  • [Cites] Pediatr Nephrol. 2007 Oct;22(10):1751-6 [17541791.001]
  • [Cites] Endocrinology. 2011 Apr;152(4):1314-26 [21266511.001]
  • [Cites] Proc Natl Acad Sci U S A. 2001 Mar 27;98(7):3756-61 [11259670.001]
  • [Cites] J Immunol. 2010 Jul 1;185(1):33-45 [20511545.001]
  • [Cites] Nat Immunol. 2004 May;5(5):495-502 [15107846.001]
  • (PMID = 28154178.001).
  • [ISSN] 1083-351X
  • [Journal-full-title] The Journal of biological chemistry
  • [ISO-abbreviation] J. Biol. Chem.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; 67-kDa laminin receptor / E3 ubiquitin ligase / EGCG / RNF216 / Toll-like receptor 4 (TLR4) / cyclic GMP (cGMP) / hypertriglyceridemia / inflammation / obesity
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8. Liu SM, Ou SY, Huang HH: Green tea polyphenols induce cell death in breast cancer MCF-7 cells through induction of cell cycle arrest and mitochondrial-mediated apoptosis. J Zhejiang Univ Sci B; 2017 Feb.;18(2):89-98

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea polyphenols induce cell death in breast cancer MCF-7 cells through induction of cell cycle arrest and mitochondrial-mediated apoptosis.
  • In order to study the molecular mechanisms of green tea polyphenols (GTPs) in treatment or prevention of breast cancer, the cytotoxic effects of GTPs on five human cell lines (MCF-7, A549, Hela, PC3, and HepG2 cells) were determined and the antitumor mechanisms of GTPs in MCF-7 cells were analyzed.

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  • [Cites] Life Sci. 2007 Jul 26;81(7):519-33 [17655876.001]
  • [Cites] Neurochem Int. 2006 Sep;49(4):379-86 [16580092.001]
  • [Cites] Carcinogenesis. 2006 Feb;27(2):262-8 [15930028.001]
  • [Cites] Eur J Pharmacol. 2012 Nov 5;694(1-3):20-9 [22939973.001]
  • [Cites] Dalton Trans. 2012 Nov 7;41(41):12766-72 [22968364.001]
  • [Cites] Apoptosis. 2006 Jun;11(6):889-904 [16547589.001]
  • [Cites] Chem Biol Interact. 2008 Oct 22;176(1):58-70 [18692031.001]
  • [Cites] Clin Cancer Res. 2008 Aug 1;14(15):4981-8 [18676773.001]
  • [Cites] J Biol Chem. 1999 Feb 19;274(8):5053-60 [9988752.001]
  • [Cites] Bioorg Med Chem Lett. 2014 Mar 15;24(6):1511-8 [24565905.001]
  • [Cites] J Zhejiang Univ Sci B. 2015 Jan;16(1):10-7 [25559951.001]
  • [Cites] Cancer Lett. 2010 Sep 28;295(2):252-9 [20359814.001]
  • [Cites] Cancer Biol Ther. 2007 Sep;6(9):1413-21 [17786034.001]
  • [Cites] Cancer Res. 2002 Feb 1;62(3):652-5 [11830514.001]
  • [Cites] J Zhejiang Univ Sci B. 2014 Jan;15(1):1-15 [24390741.001]
  • [Cites] Cancer. 1988 Dec 15;62(12):2507-16 [3056604.001]
  • [Cites] Food Chem. 2013 Nov 15;141(2):1008-18 [23790880.001]
  • [Cites] Int J Cancer. 2010 Dec 15;127(12):2893-917 [21351269.001]
  • [Cites] J Immunol. 2002 Apr 15;168(8):3902-9 [11937545.001]
  • [Cites] Oxid Med Cell Longev. 2009 Nov-Dec;2(5):297-306 [20716917.001]
  • [Cites] Toxicology. 2013 Dec 6;314(1):155-65 [24012731.001]
  • [Cites] Curr Opin Chem Biol. 2000 Feb;4(1):47-53 [10679374.001]
  • [Cites] Int J Cancer. 2010 Jun 1;126(11):2520-33 [19856314.001]
  • [Cites] Free Radic Biol Med. 2008 Jul 15;45(2):97-110 [18454943.001]
  • [Cites] Cancer Lett. 2007 Jan 8;245(1-2):232-41 [16519995.001]
  • [Cites] Annu Rev Biochem. 2000;69:217-45 [10966458.001]
  • [Cites] Arch Toxicol. 2009 Jan;83(1):11-21 [19002670.001]
  • [Cites] Food Chem Toxicol. 2012 Mar;50(3-4):1054-9 [22119783.001]
  • [Cites] J Biol Chem. 1998 Mar 6;273(10):5841-5 [9488720.001]
  • (PMID = 28124838.001).
  • [ISSN] 1862-1783
  • [Journal-full-title] Journal of Zhejiang University. Science. B
  • [ISO-abbreviation] J Zhejiang Univ Sci B
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] China
  • [Keywords] NOTNLM ; Green tea polyphenol (GTP); Breast cancer; MCF-7 cells; Mitochondrial-mediated apoptosis; Cell death; Cell cycle arrest
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9. Li W, Yalcin M, Lin Q, Ardawi MM, Mousa SA: Self-assembly of green tea catechin derivatives in nanoparticles for oral lycopene delivery. J Control Release; 2017 Feb 28;248:117-124
NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Self-assembly of green tea catechin derivatives in nanoparticles for oral lycopene delivery.
  • We selected a green tea catechin derivative, oligomerized (-)-epigallocatechin-3-O-gallate (OEGCG) as a carrier for oral lycopene delivery.

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  • [Copyright] Copyright © 2017 Elsevier B.V. All rights reserved.
  • (PMID = 28077264.001).
  • [ISSN] 1873-4995
  • [Journal-full-title] Journal of controlled release : official journal of the Controlled Release Society
  • [ISO-abbreviation] J Control Release
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
  • [Keywords] NOTNLM ; Bioavailability / Chitosan / LC-MS/MS / Lycopene / Oral delivery / Polymeric nanoparticles
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10. Green tea ingredient found to inhibit hepatitis C infection and aid liver transplantation. Nurs Stand; 2012 Jan 25;26(21):16

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea ingredient found to inhibit hepatitis C infection and aid liver transplantation.
  • : Researchers in Germany have found that green tea may have a role in preventing patients who have had a liver transplant from becoming re-infected with the hepatitis C virus.

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  • (PMID = 28051382.001).
  • [ISSN] 2047-9018
  • [Journal-full-title] Nursing standard (Royal College of Nursing (Great Britain) : 1987)
  • [ISO-abbreviation] Nurs Stand
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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