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Items 1 to 10 of about 4835
1. Nejabat M, Reza SA, Zadmehr M, Yasemi M, Sobhani Z: Efficacy of Green Tea Extract for Treatment of Dry Eye and Meibomian Gland Dysfunction; A Double-blind Randomized Controlled Clinical Trial Study. J Clin Diagn Res; 2017 Feb;11(2):NC05-NC08

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Efficacy of Green Tea Extract for Treatment of Dry Eye and Meibomian Gland Dysfunction; A Double-blind Randomized Controlled Clinical Trial Study.
  • Green tea extract has anti-oxidative, anti-bacterial, anti-androgen, and immunomodulatory properties.
  • AIM: To evaluate the efficacy of green tea extract for treatment of patients with dry eye and Meibomian Gland Dysfunction (MGD).
  • Topical green tea extract was prescribed three times a day for one month in one of the groups.
  • RESULTS: The mean age of participants in the green tea and control group was 61 and 64 years respectively.
  • In the green tea group, the mean score of clinical symptoms was 9±0.86 that improved to 4.86±0.55 after one month (p=0.002).
  • Scores suggesting improvement of TBUTs and the health of meibomian glands were significantly higher in the green tea group (p=0.002).
  • CONCLUSION: Green tea extract is an effective, safe, and well-tolerated topical treatment for mild and moderate evaporative dry eyes and MGD.

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  • [Cites] Invest Ophthalmol Vis Sci. 2011 Mar 30;52(4):1917-21 [21450912.001]
  • [Cites] Korean J Ophthalmol. 2007 Dec;21(4):232-7 [18063889.001]
  • [Cites] Obstet Gynecol. 2008 Jun;111(6):1371-9 [18515521.001]
  • [Cites] Hong Kong Med J. 2001 Dec;7(4):369-74 [11773671.001]
  • [Cites] Optom Vis Sci. 2005 Jul;82(7):594-601 [16044071.001]
  • [Cites] Korean J Ophthalmol. 2008 Sep;22(3):183-6 [18784447.001]
  • [Cites] Ocul Surf. 2016 Jul;14(3):365-76 [27224876.001]
  • [Cites] Curr Pharm Des. 2016;22(28):4470-90 [27296759.001]
  • [Cites] Arch Ophthalmol. 2000 May;118(5):615-21 [10815152.001]
  • [Cites] Mol Pharmacol. 2004 Jan;65(1):15-7 [14722232.001]
  • [Cites] Invest Ophthalmol Vis Sci. 2010 Dec;51(12):6665-70 [20702817.001]
  • [Cites] Mol Vis. 2011 Feb 18;17:533-42 [21364905.001]
  • [Cites] Ophthalmic Res. 2002 Jul-Aug;34(4):258-63 [12297700.001]
  • (PMID = 28384900.001).
  • [ISSN] 2249-782X
  • [Journal-full-title] Journal of clinical and diagnostic research : JCDR
  • [ISO-abbreviation] J Clin Diagn Res
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Keywords] NOTNLM ; Anti-bacterial / Anti-oxidative / Ocular surface / Schirmer test
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2. Chen Y, Wu Y, Du M, Chu H, Zhu L, Tong N, Zhang Z, Wang M, Gu D, Chen J: An inverse association between tea consumption and colorectal cancer risk. Oncotarget; 2017 Apr 08;

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] An inverse association between tea consumption and colorectal cancer risk.
  • It is well known that the tea extracts, mainly polyphenols as chemo-preventive elements, could act as cancer progression blockers.
  • Although the association between tea consumption and colorectal cancer risk has been widely investigated, the results still remain inconsistent.
  • The summary odds ratio (OR) of colorectal cancer for the highest vs. lowest tea consumption was 0.93 with 0.87-1.00 of 95% confidence intervals (CIs) among all studies with modest heterogeneity (P = 0.001, I2 = 43.4%).
  • Stratified analysis revealed that tea, especially green tea, had a protective effect among female and rectal cancer patients.
  • Particularly, the dose-response analysis showed that there was a significant inverse association between an increment of 1 cup/day of tea consumption and colorectal cancer risk in the subgroup of the green tea drinking (OR = 0.98, 95% CI = 0.96-1.01, Pnonlinear = 0.003) and female (OR = 0.68, 95% CI = 0.56-0.81, Pnonlinear < 0.001).
  • Our findings indicate that tea consumption has an inverse impact on colorectal cancer risk, which may have significant public health implications in the prevention of colorectal cancer and further similar researches.

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  • (PMID = 28454102.001).
  • [ISSN] 1949-2553
  • [Journal-full-title] Oncotarget
  • [ISO-abbreviation] Oncotarget
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; colorectal cancer / inverse association / tea consumption
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3. Rafieian-Kopaei M, Movahedi M: Breast cancer chemopreventive and chemotherapeutic effects of Camellia Sinensis (green tea): an updated review. Electron Physician; 2017 Feb;9(2):3838-3844
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Breast cancer chemopreventive and chemotherapeutic effects of Camellia Sinensis (green tea): an updated review.
  • This review article is aimed to overview breast cancer chemopreventive and chemotherapeutic effects of Camellia sinensis (green tea).
  • CONCLUSION: Camellia sinensis is broadly utilized as a part of customary medication since antiquated time because of its cost adequacy, and fewer reaction properties.

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  • [Cites] Cardiovasc Hematol Disord Drug Targets. 2007 Jun;7(2):135-44 [17584048.001]
  • [Cites] Pharmacology. 2014;94(5-6):245-8 [25471334.001]
  • [Cites] Front Microbiol. 2014 Aug 20;5:434 [25191312.001]
  • [Cites] Am J Clin Nutr. 2013 Dec;98(6 Suppl):1676S-1681S [24172305.001]
  • [Cites] J Hum Nutr Diet. 2015 Jun;28(3):272-82 [24646362.001]
  • [Cites] Vasc Cell. 2013 May 02;5(1):9 [23638734.001]
  • [Cites] J Nutr. 2002 Aug;132(8):2307-11 [12163680.001]
  • [Cites] J Immunol Res. 2016;2016:4962351 [27191001.001]
  • [Cites] Springerplus. 2013 May 24;2(1):240 [23750333.001]
  • [Cites] World J Clin Oncol. 2014 Aug 10;5(3):520-8 [25114865.001]
  • [Cites] RSC Adv. 2014 Aug 5;4(66):35242-35250 [25243063.001]
  • [Cites] J Evid Based Complementary Altern Med. 2015 Dec 17;:null [26682768.001]
  • [Cites] Mol Cancer. 2010 Oct 14;9:274 [20946668.001]
  • [Cites] Proc Natl Acad Sci U S A. 1999 Apr 13;96(8):4524-9 [10200295.001]
  • [Cites] World J Gastroenterol. 2013 Jun 28;19(24):3713-22 [23840110.001]
  • [Cites] J Hum Nutr Diet. 2010 Dec;23(6):590-600 [20807303.001]
  • [Cites] Arch Intern Med. 2003 Jun 23;163(12):1448-53 [12824094.001]
  • [Cites] Antiviral Res. 2003 Apr;58(2):167-73 [12742577.001]
  • [Cites] Eur J Cancer Prev. 2014 Mar;23(2):84-9 [23939462.001]
  • [Cites] Int J Surg. 2013;11(9):811-5 [23994005.001]
  • [Cites] Nutr Cancer. 2014;66(1):57-67 [24274352.001]
  • [Cites] Pharmacol Res. 2011 Aug;64(2):146-54 [21193040.001]
  • [Cites] Breast Cancer Res. 2015 Jan 31;17:15 [25637171.001]
  • [Cites] Oncol Rep. 2010 Aug;24(2):563-9 [20596647.001]
  • [Cites] J Nutr Biochem. 2014 Apr;25(4):395-403 [24561153.001]
  • [Cites] Eur J Pharmacol. 2009 Mar 15;606(1-3):172-9 [19171136.001]
  • [Cites] Cancer Epidemiol. 2016 Feb;40:73-8 [26680603.001]
  • [Cites] Nutr Res. 2013 Mar;33(3):180-7 [23507223.001]
  • [Cites] J Res Med Sci. 2014 Dec;19(12):1203-4 [25709665.001]
  • [Cites] Int J Surg. 2013;11(4):332-7 [23459184.001]
  • [Cites] Front Biosci (Elite Ed). 2012 Jan 01;4:111-31 [22201858.001]
  • [Cites] J Renal Inj Prev. 2013 Nov 09;3(2):47-9 [25340167.001]
  • [Cites] Phytomedicine. 2010 Jan;17(1):3-13 [19959000.001]
  • [Cites] Int J Cancer. 2009 Mar 15;124(6):1404-8 [19048616.001]
  • [Cites] Crit Rev Food Sci Nutr. 1997 Dec;37(8):693-704 [9447270.001]
  • [Cites] Carcinogenesis. 2008 Oct;29(10):1967-72 [18669903.001]
  • [Cites] Front Oncol. 2013 Dec 13;3:298 [24380073.001]
  • [Cites] Iran J Public Health. 2014 Sep;43(9):1311-3 [26175992.001]
  • [Cites] J Evid Based Complementary Altern Med. 2016 Sep 11;:null [27620926.001]
  • [Cites] Curr Pharm Des. 2016;22(3):277-85 [26561063.001]
  • [Cites] Arch Biochem Biophys. 2010 Sep 1;501(1):65-72 [20558130.001]
  • [Cites] Breast Cancer Res Treat. 2015 Feb;149(3):655-68 [25663548.001]
  • [Cites] Breast Cancer Res. 2010;12 (5):R88 [22889409.001]
  • [Cites] Oxid Med Cell Longev. 2012;2012:984219 [22666523.001]
  • [Cites] Am J Clin Nutr. 2000 Jun;71(6 Suppl):1698S-702S; discussion 1703S-4S [10837321.001]
  • [Cites] Curr Pharm Des. 2016;22(2):238-46 [26561062.001]
  • [Cites] Ann N Y Acad Sci. 2001 Apr;928:274-80 [11795518.001]
  • [Cites] PLoS One. 2014 Jan 30;9(1):e87580 [24498143.001]
  • [Cites] Iran J Kidney Dis. 2015 Nov;9(6):421-6 [26552347.001]
  • [Cites] J Evid Based Complementary Altern Med. 2016 Jul;21(3):221-7 [26232244.001]
  • [Cites] J Renal Inj Prev. 2016 Aug 09;5(3):123-8 [27689107.001]
  • [Cites] Cancer Genomics Proteomics. 2010 Sep-Oct;7(5):261-77 [20952761.001]
  • [Cites] Integr Cancer Ther. 2005 Jun;4(2):144-55 [15911927.001]
  • [Cites] Bioorg Med Chem. 2010 Feb;18(3):1252-8 [20045338.001]
  • [Cites] Mol Nutr Food Res. 2011 Jun;55(6):844-54 [21538850.001]
  • [Cites] Food Chem Toxicol. 2015 Sep;83:26-35 [26051348.001]
  • [Cites] Cancer Causes Control. 2015 Oct;26(10 ):1405-19 [26206423.001]
  • [Cites] J Nutr. 2009 Feb;139(2):310-6 [19074205.001]
  • [Cites] PLoS One. 2014 May 14;9(5):e96013 [24828424.001]
  • [Cites] Iran J Basic Med Sci. 2015 Oct;18(10):967-73 [26730330.001]
  • [Cites] Int J Cancer. 2015 Oct 15;137(8):1979-89 [25885188.001]
  • [Cites] Biomed Res Int. 2013;2013:412379 [24102055.001]
  • [Cites] Crit Rev Food Sci Nutr. 2003;43(1):89-143 [12587987.001]
  • [Cites] Res Pharm Sci. 2016 Mar-Apr;11(2):145-51 [27168754.001]
  • [Cites] Acta Biomed. 2015 Sep 14;86(2):130-6 [26422426.001]
  • [Cites] Int J Antimicrob Agents. 2009 May;33(5):473-8 [19157800.001]
  • (PMID = 28465816.001).
  • [Journal-full-title] Electronic physician
  • [ISO-abbreviation] Electron Physician
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Iran
  • [Keywords] NOTNLM ; Camellia sinensis / Phytochemicals / catechins / chemopreventive / chemotherapeutic
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4. Li X, Xu K, Zhang Y, Sun C, He Y: Optical Determination of Lead Chrome Green in Green Tea by Fourier Transform Infrared (FT-IR) Transmission Spectroscopy. PLoS One; 2017;12(1):e0169430

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Optical Determination of Lead Chrome Green in Green Tea by Fourier Transform Infrared (FT-IR) Transmission Spectroscopy.
  • The potential of Fourier transform infrared (FT-IR) transmission spectroscopy for determination of lead chrome green in green tea was investigated based on chemometric methods.
  • Firstly, the qualitative analysis of lead chrome green in tea was performed based on partial least squares discriminant analysis (PLS-DA), and the correct rate of classification was 100%.
  • And then, a hybrid method of interval partial least squares (iPLS) regression and successive projections algorithm (SPA) was proposed to select characteristic wavenumbers for the quantitative analysis of lead chrome green in green tea, and 19 wavenumbers were obtained finally.
  • Among these wavenumbers, 1384 (C = C), 1456, 1438, 1419(C = N), and 1506 (CNH) cm-1 were the characteristic wavenumbers of lead chrome green.
  • All these results indicated the feasibility of IR spectra for detecting lead chrome green in green tea.

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  • [Cites] Food Chem. 2015 Sep 15;183:30-5 [25863606.001]
  • [Cites] Spectrochim Acta A Mol Biomol Spectrosc. 2013 Jul;111:31-6 [23602956.001]
  • [Cites] Food Chem. 2015 Jul 15;179:175-81 [25722152.001]
  • [Cites] Food Chem. 2014 Sep 1;158:351-7 [24731354.001]
  • [Cites] Anal Chim Acta. 2008 May 12;615(1):10-7 [18440358.001]
  • [Cites] Sci Rep. 2015 Oct 28;5:15729 [26508516.001]
  • [Cites] Anal Chim Acta. 2009 Apr 6;638(1):16-22 [19298874.001]
  • [Cites] Food Chem. 2015 Jun 1;176:130-6 [25624215.001]
  • [Cites] Neural Netw. 2001 Jan;14(1):23-35 [11213211.001]
  • [Cites] Biol Trace Elem Res. 2010 Aug;136(2):127-39 [20195925.001]
  • [Cites] Food Chem Toxicol. 2014 Mar;65:227-32 [24394486.001]
  • [Cites] Appl Spectrosc. 2007 Mar;61(3):293-9 [17389069.001]
  • [Cites] Planta Med. 2014 Aug;80(12):1023-8 [25098931.001]
  • (PMID = 28068348.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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5. Rashidi B, Malekzadeh M, Goodarzi M, Masoudifar A, Mirzaei H: Green tea and its anti-angiogenesis effects. Biomed Pharmacother; 2017 May;89:949-956

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea and its anti-angiogenesis effects.
  • Green tea (from the Camellia sinensis plant) is one of the most popular beverages in the world.
  • Green tea and its polyphenolic substances (like catechins) show chemo-preventive and chemotherapeutic features in various types of cancer and experimental models for human cancers.
  • The tea catechins, including (-)-epigallocatechin-3-gallate (EGCG), have multiple effects on the cellular proteome and signalome.
  • Note that the polyphenolic compounds from green tea are able to change the miRNA expression profile associated with angiogenesis in various cancer types.
  • This review focuses on the ability of the green tea constituents to suppress angiogenesis signaling and it summarizes the mechanisms by which EGCG might inhibit the VEGF family.
  • We also highlighted the miRNAs affected by green tea which are involved in anti-angiogenesis.

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  • [Copyright] Copyright © 2017 Elsevier Masson SAS. All rights reserved.
  • (PMID = 28292023.001).
  • [ISSN] 1950-6007
  • [Journal-full-title] Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
  • [ISO-abbreviation] Biomed. Pharmacother.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] France
  • [Keywords] NOTNLM ; Angiogenesis / Epigallocatechin-3-gallate / Green tea / MicroRNA / VEGF
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6. Maiti S, Acharyya N, Ghosh TK, Ali SS, Manna E, Nazmeen A, Sinha NK: Green Tea (Camellia sinensis) Protects Against Arsenic Neurotoxicity via Antioxidative Mechanism And Activation of Superoxide Dismutase Activity. Cent Nerv Syst Agents Med Chem; 2017 Feb 01;

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea (Camellia sinensis) Protects Against Arsenic Neurotoxicity via Antioxidative Mechanism And Activation of Superoxide Dismutase Activity.
  • Here, the protective role of Green tea (Camellia sinensis or CS; 10mg/ml aqueous) has been evaluated against arsenic-induced (0.6ppm/100g bw/28 days) cerebral/cerebellar tissue degeneration, oxidative-threats and neurotransmitter deregulation in female rats.

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  • (PMID = 28155600.001).
  • [ISSN] 1875-6166
  • [Journal-full-title] Central nervous system agents in medicinal chemistry
  • [ISO-abbreviation] Cent Nerv Syst Agents Med Chem
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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7. Li J, Sapper TN, Mah E, Moller MV, Kim JB, Chitchumroonchokchai C, McDonald JD, Bruno RS: Green tea extract treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by lowering TNFR1 and TLR4 expression and ligand availability. J Nutr Biochem; 2017 Mar;41:34-41
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea extract treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by lowering TNFR1 and TLR4 expression and ligand availability.
  • We hypothesized that antiinflammatory activities of green tea extract (GTE) during NASH would lower tumor necrosis factor receptor-1 (TNFR1)- and Toll-like receptor-4 (TLR4)-mediated NFκB activation.
  • These data suggest that dietary GTE treatment reduces hepatic inflammation in NASH by decreasing proinflammatory signaling through TNFR1 and TLR4 that otherwise increases NFκB activation and liver injury.

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  • [Copyright] Copyright © 2016 Elsevier Inc. All rights reserved.
  • (PMID = 28038359.001).
  • [ISSN] 1873-4847
  • [Journal-full-title] The Journal of nutritional biochemistry
  • [ISO-abbreviation] J. Nutr. Biochem.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Green tea / Inflammation / NASH / Nonalcoholic steatohepatitis / TLR4 / TNFR1
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8. Farhan M, Oves M, Chibber S, Hadi SM, Ahmad A: Mobilization of Nuclear Copper by Green Tea Polyphenol Epicatechin-3-Gallate and Subsequent Prooxidant Breakage of Cellular DNA: Implications for Cancer Chemotherapy. Int J Mol Sci; 2016 Dec 26;18(1)
Hazardous Substances Data Bank. COPPER, ELEMENTAL .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mobilization of Nuclear Copper by Green Tea Polyphenol Epicatechin-3-Gallate and Subsequent Prooxidant Breakage of Cellular DNA: Implications for Cancer Chemotherapy.
  • Epidemiological as well as experimental evidence exists in support of chemopreventive and anticancer properties of green tea and its constituents.
  • The gallocatechin, epicatechin-3-gallate is a major polyphenol present in green tea, shown responsible for these effects.
  • Our results are indicative of ROS generation, possibly through mobilization of endogenous copper ions, and support our long-standing hypothesis of a prooxidant activity of plant-derived polyphenols as a mechanism for their documented anticancer properties.
  • [MeSH-minor] Cell Line, Tumor. Cells, Cultured. Humans. Lymphocytes / drug effects. Lymphocytes / metabolism. Reactive Oxygen Species / metabolism. Tea / chemistry

  • Hazardous Substances Data Bank. Green tea .
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  • [Cites] Cancer Lett. 2000 Jun 1;154(1):29-37 [10799736.001]
  • [Cites] Biochem Biophys Res Commun. 1984 Aug 30;123(1):291-8 [6477583.001]
  • [Cites] Annu Rev Biochem. 1980;49:695-726 [6250449.001]
  • [Cites] Free Radic Biol Med. 1994 Jan;16(1):43-8 [8299995.001]
  • [Cites] Food Chem Toxicol. 1993 Apr;31(4):271-83 [7682977.001]
  • [Cites] Biochem J. 1997 Apr 15;323 ( Pt 2):337-41 [9163321.001]
  • [Cites] J Biol Chem. 2010 Nov 5;285(45):34557-65 [20826787.001]
  • [Cites] J Natl Cancer Inst. 1997 Dec 17;89(24):1881-6 [9414176.001]
  • [Cites] Stem Cells. 1995 May;13 Suppl 1:207-14 [7488947.001]
  • [Cites] Clin Cancer Res. 2011 Aug 15;17(16):5402-11 [21705453.001]
  • [Cites] Blood. 1998 Aug 1;92(3):996-1002 [9680369.001]
  • [Cites] Mutat Res. 1994 Aug;313(1):39-48 [7519309.001]
  • [Cites] FEBS Lett. 2006 Jan 23;580(2):533-8 [16412432.001]
  • [Cites] Cancer Lett. 1998 Jul 17;129(2):173-9 [9719459.001]
  • [Cites] Open Biol. 2013 Jan 08;3(1):120144 [23303309.001]
  • [Cites] J Oncol. 2012;2012:749310 [23316231.001]
  • [Cites] Biochim Biophys Acta. 1996 Nov 15;1317(2):95-100 [8950193.001]
  • [Cites] Mutagenesis. 1998 May;13(3):271-4 [9643586.001]
  • [Cites] J Biol Chem. 1991 Oct 25;266(30):20175-84 [1939078.001]
  • [Cites] Toxicology. 2006 Aug 15;225(2-3):173-82 [16843582.001]
  • [Cites] Free Radic Biol Med. 2002 Aug 1;33(3):387-98 [12126761.001]
  • [Cites] Mol Nutr Food Res. 2011 Apr;55(4):553-9 [21462322.001]
  • [Cites] Cancer Cell. 2006 Sep;10(3):241-52 [16959615.001]
  • [Cites] Free Radic Biol Med. 1999 Sep;27(5-6):612-6 [10490282.001]
  • [Cites] FEBS Lett. 2005 Jun 6;579(14 ):3131-5 [15919081.001]
  • [Cites] Biochem Biophys Res Commun. 2000 Jun 24;273(1):50-3 [10873562.001]
  • [Cites] IUBMB Life. 2000 Sep;50(3):167-71 [11142343.001]
  • [Cites] Free Radic Biol Med. 1994 Jun;16(6):845-50 [8070690.001]
  • [Cites] Mol Nutr Food Res. 2014 Mar;58(3):437-46 [24123728.001]
  • [Cites] Nature. 2012 Apr 18;486(7403):346-52 [22522925.001]
  • [Cites] Cancer Res. 1988 May 1;48(9):2361-5 [3128399.001]
  • [Cites] Biochem Biophys Res Commun. 1994 Oct 28;204(2):898-904 [7980558.001]
  • [Cites] Carcinogenesis. 1992 Apr;13(4):605-8 [1315626.001]
  • [Cites] Biochem Pharmacol. 1987 Nov 1;36(21):3629-33 [2823829.001]
  • [Cites] Cancer Lett. 1998 Feb 13;124(1):23-30 [9500187.001]
  • [Cites] Food Chem Toxicol. 1992 Jun;30(6):483-9 [1379971.001]
  • [Cites] Free Radic Res. 2008 Aug;42(8):764-72 [18661438.001]
  • [Cites] Phytother Res. 2003 Apr;17(4):358-63 [12722140.001]
  • [Cites] Cancer Cell. 2006 Sep;10(3):175-6 [16959608.001]
  • (PMID = 28035959.001).
  • [ISSN] 1422-0067
  • [Journal-full-title] International journal of molecular sciences
  • [ISO-abbreviation] Int J Mol Sci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Antineoplastic Agents, Phytogenic; 0 / Reactive Oxygen Species; 0 / Tea; 789U1901C5 / Copper; 8R1V1STN48 / Catechin; 92587OVD8Z / epicatechin gallate
  • [Keywords] NOTNLM ; DNA damage (major topic) / anticancer (major topic) / comet assay (major topic) / copper (major topic) / epicatechin-3-gallate (major topic) / prooxidant (major topic) / reactive oxygen species (major topic)
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9. Li W, Yalcin M, Lin Q, Ardawi MM, Mousa SA: Self-assembly of green tea catechin derivatives in nanoparticles for oral lycopene delivery. J Control Release; 2017 Feb 28;248:117-124
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Self-assembly of green tea catechin derivatives in nanoparticles for oral lycopene delivery.
  • We selected a green tea catechin derivative, oligomerized (-)-epigallocatechin-3-O-gallate (OEGCG) as a carrier for oral lycopene delivery.

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  • [Copyright] Copyright © 2017 Elsevier B.V. All rights reserved.
  • (PMID = 28077264.001).
  • [ISSN] 1873-4995
  • [Journal-full-title] Journal of controlled release : official journal of the Controlled Release Society
  • [ISO-abbreviation] J Control Release
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
  • [Keywords] NOTNLM ; Bioavailability / Chitosan / LC-MS/MS / Lycopene / Oral delivery / Polymeric nanoparticles
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10. Xinqiang S, Mu Z, Lei C, Mun LY: Bioinformatics Analysis on Molecular Mechanism of Green tea Compound Epigallocatechin-3-Gallate Against Ovarian Cancer. Clin Transl Sci; 2017 May 15;
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Bioinformatics Analysis on Molecular Mechanism of Green tea Compound Epigallocatechin-3-Gallate Against Ovarian Cancer.
  • Epigallocatechin-3-gallate (EGCG) is the most abundant and biologically active catechin in green tea, and it exerts multiple effects in humans through mechanisms that remain to be clarified.

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
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  • [Copyright] This article is protected by copyright. All rights reserved.
  • (PMID = 28504421.001).
  • [ISSN] 1752-8062
  • [Journal-full-title] Clinical and translational science
  • [ISO-abbreviation] Clin Transl Sci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Ingenuity Pathway Analysis / bioinformatics analysis / epigallocatechin-3-gallate / ovarian cancer
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