Find all
associated with


Refine your query (more in Advanced-Search):
 Focus on the recent 5 years   Focus on the current year   Focus on the last 30 days   More choices ...
 Focus on articles with free fulltexts   More choices ...
 Do simple 'keyword' search (no query expansion)

[X] Close
You are about to erase all the values you have customized, search history, page format, etc.
Click here to RESET all values       Click here to GO BACK without resetting any value
Items 1 to 10 of about 5003
1. Li X, Xu K, Zhang Y, Sun C, He Y: Optical Determination of Lead Chrome Green in Green Tea by Fourier Transform Infrared (FT-IR) Transmission Spectroscopy. PLoS One; 2017;12(1):e0169430
Hazardous Substances Data Bank. Lead, elemental .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Optical Determination of Lead Chrome Green in Green Tea by Fourier Transform Infrared (FT-IR) Transmission Spectroscopy.
  • The potential of Fourier transform infrared (FT-IR) transmission spectroscopy for determination of lead chrome green in green tea was investigated based on chemometric methods.
  • Firstly, the qualitative analysis of lead chrome green in tea was performed based on partial least squares discriminant analysis (PLS-DA), and the correct rate of classification was 100%.
  • And then, a hybrid method of interval partial least squares (iPLS) regression and successive projections algorithm (SPA) was proposed to select characteristic wavenumbers for the quantitative analysis of lead chrome green in green tea, and 19 wavenumbers were obtained finally.
  • Among these wavenumbers, 1384 (C = C), 1456, 1438, 1419(C = N), and 1506 (CNH) cm-1 were the characteristic wavenumbers of lead chrome green.
  • All these results indicated the feasibility of IR spectra for detecting lead chrome green in green tea.
  • [MeSH-major] Coloring Agents / analysis. Lead / analysis. Spectroscopy, Fourier Transform Infrared. Tea / chemistry

  • Hazardous Substances Data Bank. Green tea .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Food Chem. 2015 Sep 15;183:30-5 [25863606.001]
  • [Cites] Spectrochim Acta A Mol Biomol Spectrosc. 2013 Jul;111:31-6 [23602956.001]
  • [Cites] Food Chem. 2015 Jul 15;179:175-81 [25722152.001]
  • [Cites] Food Chem. 2014 Sep 1;158:351-7 [24731354.001]
  • [Cites] Anal Chim Acta. 2008 May 12;615(1):10-7 [18440358.001]
  • [Cites] Sci Rep. 2015 Oct 28;5:15729 [26508516.001]
  • [Cites] Anal Chim Acta. 2009 Apr 6;638(1):16-22 [19298874.001]
  • [Cites] Food Chem. 2015 Jun 1;176:130-6 [25624215.001]
  • [Cites] Neural Netw. 2001 Jan;14(1):23-35 [11213211.001]
  • [Cites] Biol Trace Elem Res. 2010 Aug;136(2):127-39 [20195925.001]
  • [Cites] Food Chem Toxicol. 2014 Mar;65:227-32 [24394486.001]
  • [Cites] Appl Spectrosc. 2007 Mar;61(3):293-9 [17389069.001]
  • [Cites] Planta Med. 2014 Aug;80(12):1023-8 [25098931.001]
  • (PMID = 28068348.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Coloring Agents; 0 / Tea; 2P299V784P / Lead
  •  go-up   go-down


2. Park JH, Bae JH, Im SS, Song DK: Green tea and type 2 diabetes. Integr Med Res; 2014 Mar;3(1):4-10

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea and type 2 diabetes.
  • Green tea and coffee consumption have been widely popular worldwide.
  • In addition to this caffeine effect, green tea and coffee consumption have always been at the center of discussions about human health, disease, and longevity.
  • In particular, green tea catechins are involved in many biological activities such as antioxidation and modulation of various cellular lipid and proteins.
  • Some reports also suggest that daily consumption of tea catechins may help in controlling type 2 diabetes.
  • However, other studies have reported that chronic consumption of green tea may result in hepatic failure, neuronal damage, and exacerbation of diabetes, suggesting that interindividual variations in the green tea effect are large.
  • This review will focus on the effect of green tea catechins extracted from the <i>Camellia sinensis</i> plant on type 2 diabetes and obesity, and the possible mechanistic explanation for the experimental results mainly from our laboratory.
  • It is hoped that green tea can be consumed in a suitable manner as a supplement to prevent the development of type 2 diabetes and obesity.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Eur J Pharmacol. 2006 Jul 10;541(1-2):115-21 [16765345.001]
  • [Cites] Free Radic Biol Med. 2005 Sep 15;39(6):752-61 [16109305.001]
  • [Cites] FEBS Lett. 2005 Mar 14;579(7):1653-7 [15757656.001]
  • [Cites] Biochem Pharmacol. 2005 Nov 25;70(11):1560-7 [16216226.001]
  • [Cites] Biochem J. 2005 Mar 15;386(Pt 3):471-8 [15469417.001]
  • [Cites] J Antimicrob Chemother. 2004 Feb;53(2):225-9 [14688042.001]
  • [Cites] Biochem Biophys Res Commun. 1999 Apr 2;257(1):79-83 [10092513.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2001 Jan;10(1):53-8 [11205489.001]
  • [Cites] Naunyn Schmiedebergs Arch Pharmacol. 2010 Oct;382(4):303-10 [20711765.001]
  • [Cites] J Physiol Pharmacol. 2009 Dec;60(4):101-9 [20065503.001]
  • [Cites] Obesity (Silver Spring). 2009 Feb;17(2):310-7 [19008868.001]
  • [Cites] Arch Biochem Biophys. 1995 Oct 1;322(2):339-46 [7574706.001]
  • [Cites] Obes Res. 2003 Sep;11(9):1088-95 [12972679.001]
  • [Cites] Altern Med Rev. 2011 Jun;16(2):157-63 [21649457.001]
  • [Cites] Obes Res. 2005 Jun;13(6):982-90 [15976140.001]
  • [Cites] Pharmacol Res. 2007 Sep;56(3):237-47 [17656102.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2002 Oct;11(10 Pt 1):1025-32 [12376503.001]
  • [Cites] Biol Pharm Bull. 2008 Jul;31(7):1403-9 [18591783.001]
  • [Cites] J Nutr. 2003 Oct;133(10):3262S-3267S [14519824.001]
  • [Cites] Biosci Biotechnol Biochem. 1997 Dec;61(12 ):1981-5 [9438978.001]
  • [Cites] Biochem Pharmacol. 2001 Sep 1;62(5):527-35 [11585049.001]
  • [Cites] Horm Metab Res. 2007 Oct;39(10):717-21 [17952832.001]
  • [Cites] Metabolism. 2007 Oct;56(10):1340-4 [17884442.001]
  • [Cites] J Agric Food Chem. 2009 Aug 12;57(15):6685-91 [19601628.001]
  • [Cites] Biosci Biotechnol Biochem. 2001 Dec;65(12):2638-43 [11826958.001]
  • [Cites] Br J Pharmacol. 2003 Oct;140(3):487-99 [12970085.001]
  • [Cites] Drug Metab Dispos. 1997 Sep;25(9):1045-50 [9311619.001]
  • [Cites] J Agric Food Chem. 2002 Nov 20;50(24):7182-6 [12428980.001]
  • [Cites] Ann Intern Med. 2006 Apr 18;144(8):554-62 [16618952.001]
  • [Cites] Mol Nutr Food Res. 2009 Mar;53(3):349-60 [19065584.001]
  • [Cites] Obesity (Silver Spring). 2007 Nov;15(11):2571-82 [18070748.001]
  • [Cites] Arterioscler Thromb Vasc Biol. 2004 Jan;24(1):29-33 [14551151.001]
  • [Cites] Cell Biol Int. 2007 Nov;31(11):1379-87 [17631393.001]
  • [Cites] J Nutr Biochem. 2003 Jun;14(6):326-32 [12873714.001]
  • [Cites] Mol Biochem Parasitol. 2009 Nov;168(1):113-6 [19577593.001]
  • [Cites] Am J Physiol Cell Physiol. 2005 May;288(5):C1094-108 [15647388.001]
  • [Cites] Diabetes Res Clin Pract. 2006 Mar;71(3):356-8 [16169629.001]
  • [Cites] J Agric Food Chem. 2000 Nov;48(11):5618-23 [11087528.001]
  • [Cites] J Agric Food Chem. 2005 Jul 13;53(14):5695-701 [15998135.001]
  • [Cites] J Nutr Sci Vitaminol (Tokyo). 2005 Oct;51(5):335-42 [16392704.001]
  • [Cites] Nature. 2005 Jul 21;436(7049):356-62 [16034410.001]
  • [Cites] Free Radic Res. 2003 Aug;37(8):881-90 [14567448.001]
  • [Cites] Naunyn Schmiedebergs Arch Pharmacol. 2013 Aug;386(8):733-45 [23620335.001]
  • [Cites] Diabetes Metab J. 2013 Jun;37(3):196-206 [23807923.001]
  • [Cites] Naunyn Schmiedebergs Arch Pharmacol. 2009 Jun;379(6):551-64 [19221718.001]
  • [Cites] J Clin Invest. 2005 Aug;115(8):2047-58 [16075046.001]
  • [Cites] Nutrition. 2009 Oct;25(10):1047-56 [19535224.001]
  • [Cites] J Agric Food Chem. 2000 Oct;48(10):4576-80 [11052703.001]
  • (PMID = 28664072.001).
  • [ISSN] 2213-4220
  • [Journal-full-title] Integrative medicine research
  • [ISO-abbreviation] Integr Med Res
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Netherlands
  • [Keywords] NOTNLM ; catechins / glucose uptake / green tea / obesity / type 2 diabetes
  •  go-up   go-down


3. Nejabat M, Reza SA, Zadmehr M, Yasemi M, Sobhani Z: Efficacy of Green Tea Extract for Treatment of Dry Eye and Meibomian Gland Dysfunction; A Double-blind Randomized Controlled Clinical Trial Study. J Clin Diagn Res; 2017 Feb;11(2):NC05-NC08

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Efficacy of Green Tea Extract for Treatment of Dry Eye and Meibomian Gland Dysfunction; A Double-blind Randomized Controlled Clinical Trial Study.
  • Green tea extract has anti-oxidative, anti-bacterial, anti-androgen, and immunomodulatory properties.
  • AIM: To evaluate the efficacy of green tea extract for treatment of patients with dry eye and Meibomian Gland Dysfunction (MGD).
  • Topical green tea extract was prescribed three times a day for one month in one of the groups.
  • RESULTS: The mean age of participants in the green tea and control group was 61 and 64 years respectively.
  • In the green tea group, the mean score of clinical symptoms was 9±0.86 that improved to 4.86±0.55 after one month (p=0.002).
  • Scores suggesting improvement of TBUTs and the health of meibomian glands were significantly higher in the green tea group (p=0.002).
  • CONCLUSION: Green tea extract is an effective, safe, and well-tolerated topical treatment for mild and moderate evaporative dry eyes and MGD.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Invest Ophthalmol Vis Sci. 2011 Mar 30;52(4):1917-21 [21450912.001]
  • [Cites] Korean J Ophthalmol. 2007 Dec;21(4):232-7 [18063889.001]
  • [Cites] Obstet Gynecol. 2008 Jun;111(6):1371-9 [18515521.001]
  • [Cites] Hong Kong Med J. 2001 Dec;7(4):369-74 [11773671.001]
  • [Cites] Optom Vis Sci. 2005 Jul;82(7):594-601 [16044071.001]
  • [Cites] Korean J Ophthalmol. 2008 Sep;22(3):183-6 [18784447.001]
  • [Cites] Ocul Surf. 2016 Jul;14(3):365-76 [27224876.001]
  • [Cites] Curr Pharm Des. 2016;22(28):4470-90 [27296759.001]
  • [Cites] Arch Ophthalmol. 2000 May;118(5):615-21 [10815152.001]
  • [Cites] Mol Pharmacol. 2004 Jan;65(1):15-7 [14722232.001]
  • [Cites] Invest Ophthalmol Vis Sci. 2010 Dec;51(12):6665-70 [20702817.001]
  • [Cites] Mol Vis. 2011 Feb 18;17:533-42 [21364905.001]
  • [Cites] Ophthalmic Res. 2002 Jul-Aug;34(4):258-63 [12297700.001]
  • (PMID = 28384900.001).
  • [ISSN] 2249-782X
  • [Journal-full-title] Journal of clinical and diagnostic research : JCDR
  • [ISO-abbreviation] J Clin Diagn Res
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Keywords] NOTNLM ; Anti-bacterial / Anti-oxidative / Ocular surface / Schirmer test
  •  go-up   go-down


Advertisement
4. Matsuo T, Miyata Y, Asai A, Sagara Y, Furusato B, Fukuoka J, Sakai H: Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer. PLoS One; 2017;12(1):e0171091
NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer.
  • Green tea polyphenol (GTP) suppresses carcinogenesis and aggressiveness in many types of malignancies including bladder cancer.
  • [MeSH-major] Polyphenols / pharmacology. Tea / chemistry. Urinary Bladder Neoplasms / metabolism

  • MedlinePlus Health Information. consumer health - Bladder Cancer.
  • Hazardous Substances Data Bank. Green tea .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Life Sci. 2013 Sep 3;93(8):307-12 [23871988.001]
  • [Cites] DNA Cell Biol. 2003 Mar;22(3):217-24 [12804120.001]
  • [Cites] J Biol Chem. 2006 Nov 3;281(44):33761-72 [16950787.001]
  • [Cites] PLoS One. 2013;8(3):e59095 [23516604.001]
  • [Cites] Cancer Res. 2006 Jan 15;66(2):1234-40 [16424063.001]
  • [Cites] Vasc Cell. 2013 May 02;5(1):9 [23638734.001]
  • [Cites] J Nutr. 2002 Aug;132(8):2307-11 [12163680.001]
  • [Cites] Urol Int. 2013;90(1):10-6 [23052791.001]
  • [Cites] Oncotarget. 2015 Sep 29;6(29):27312-31 [26314962.001]
  • [Cites] Cancer Epidemiol. 2010 Jun;34(3):350-4 [20362526.001]
  • [Cites] Carcinogenesis. 2004 Nov;25(11):2217-24 [15358631.001]
  • [Cites] Biochem Pharmacol. 2011 Dec 15;82(12):1807-21 [21827739.001]
  • [Cites] Br J Cancer. 2001 Mar 23;84(6):844-50 [11259102.001]
  • [Cites] Clin Cancer Res. 2003 May;9(5):1741-9 [12738729.001]
  • [Cites] Clin Cancer Res. 2007 Dec 1;13(23):6959-63 [18056170.001]
  • [Cites] Urology. 2011 Aug;78(2):476.e9-15 [21696810.001]
  • [Cites] Cancer Lett. 2001 Jun 26;167(2):175-82 [11369139.001]
  • [Cites] Mol Cell Biol. 2009 May;29(10 ):2622-35 [19289500.001]
  • [Cites] Cancer Res Treat. 2009 Jun;41(2):87-92 [19707506.001]
  • [Cites] Cancer Epidemiol. 2015 Dec;39 Suppl 1:S84-92 [26164655.001]
  • [Cites] Rev Urol. 2006 Winter;8(1):8-13 [16985555.001]
  • [Cites] Biochem Biophys Res Commun. 2007 Mar 23;354(4):852-7 [17266926.001]
  • [Cites] Sci Rep. 2015 Mar 12;5:9051 [25761588.001]
  • [Cites] Tumour Biol. 2015 Jan;36(1):315-27 [25252849.001]
  • [Cites] Biomed Res. 2009 Aug;30(4):207-15 [19729851.001]
  • [Cites] Basic Clin Pharmacol Toxicol. 2015 Sep;117(3):164-72 [25625183.001]
  • [Cites] Transl Res. 2014 Dec;164(6):468-76 [25063314.001]
  • [Cites] Clin Nutr. 2013 Dec;32(6):894-903 [23582951.001]
  • [Cites] Anticancer Res. 2014 Feb;34(2):735-44 [24511007.001]
  • [Cites] J Natl Cancer Inst. 2007 Apr 4;99(7):558-68 [17406000.001]
  • [Cites] Life Sci. 2006 Nov 17;79(25):2329-36 [16945390.001]
  • [Cites] J Neurooncol. 2008 Jun;88(2):143-55 [18317686.001]
  • [Cites] Biochem Biophys Res Commun. 2012 Nov 2;427(4):725-30 [23036202.001]
  • [Cites] Med Oncol. 2011 Dec;28 Suppl 1:S577-85 [21046284.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2015 Oct;24(10):1516-22 [26215293.001]
  • [Cites] Am J Transl Res. 2016 Feb 15;8(2):578-87 [27158349.001]
  • [Cites] Clin Cancer Res. 2006 Feb 1;12(3 Pt 1):800-6 [16467091.001]
  • [Cites] Tumour Biol. 2016 Apr;37(4):4373-82 [26499783.001]
  • [Cites] Mol Cancer Res. 2011 May;9(5):648-59 [21498545.001]
  • [Cites] PLoS One. 2011;6(10):e25224 [22022384.001]
  • [Cites] Nutr Cancer. 2014;66(2):315-24 [24447094.001]
  • [Cites] J Gastroenterol. 2014 Apr;49(4):692-701 [23543313.001]
  • [Cites] Oncol Rep. 2015 Jun;33(6):2972-80 [25845434.001]
  • [Cites] Int J Cancer. 1997 Jan 27;70(3):255-8 [9033623.001]
  • [Cites] Leuk Res. 2007 Sep;31(9):1277-84 [17081606.001]
  • (PMID = 28141864.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / ELAV-Like Protein 1; 0 / Polyphenols; 0 / Tea
  •  go-up   go-down


5. Xinqiang S, Mu Z, Lei C, Mun LY: Bioinformatics Analysis on Molecular Mechanism of Green Tea Compound Epigallocatechin-3-Gallate Against Ovarian Cancer. Clin Transl Sci; 2017 Jul;10(4):302-307
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Bioinformatics Analysis on Molecular Mechanism of Green Tea Compound Epigallocatechin-3-Gallate Against Ovarian Cancer.
  • Epigallocatechin-3-gallate (EGCG) is the most abundant and biologically active catechin in green tea, and it exerts multiple effects in humans through mechanisms that remain to be clarified.

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] © 2017 The Authors. Clinical and Translational Science published by Wiley Periodicals, Inc. on behalf of American Society for Clinical Pharmacology and Therapeutics.
  • [Cites] Exp Biol Med (Maywood). 2006 Jun;231(6):1123-7 [16741061.001]
  • [Cites] Nutrients. 2016 Sep 09;8(9): [27618095.001]
  • [Cites] Lancet. 2005 Jan 8-14;365(9454):101-2 [15639276.001]
  • [Cites] Lancet. 1997 Jan 11;349(9045):113-7 [8996432.001]
  • [Cites] Arch Intern Med. 2005 Dec 12-26;165(22):2683-6 [16344429.001]
  • [Cites] Carcinogenesis. 1998 Apr;19(4):611-6 [9600345.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2002 Aug;11(8):713-8 [12163323.001]
  • [Cites] Cancer Res. 2006 Mar 1;66(5):2500-5 [16510563.001]
  • [Cites] Anticancer Res. 2011 Apr;31(4):1131-40 [21508356.001]
  • [Cites] Front Biosci (Elite Ed). 2012 Jan 01;4:111-31 [22201858.001]
  • [Cites] Cancer Epidemiol. 2013 Feb;37(1):54-9 [23107758.001]
  • [Cites] Am J Clin Nutr. 2012 May;95(5):1172-81 [22440851.001]
  • [Cites] Mol Cancer Ther. 2006 Jun;5(6):1483-92 [16818507.001]
  • [Cites] Nutr Cancer. 2011;63(6):817-26 [21800977.001]
  • [Cites] J Agric Food Chem. 2003 Jul 16;51(15):4427-35 [12848521.001]
  • [Cites] Arch Pharm Res. 2000 Dec;23(6):605-12 [11156183.001]
  • [Cites] Evid Based Complement Alternat Med. 2009 Dec;6(4):523-30 [18955299.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2008 Mar;17 (3):712-6 [18349292.001]
  • [Cites] Oncol Rep. 2010 Mar;23(3):605-14 [20126997.001]
  • [Cites] Gynecol Oncol. 2012 Sep;126(3):491-8 [22564714.001]
  • [Cites] Nutrients. 2012 Nov 08;4(11):1679-91 [23201840.001]
  • [Cites] Genes Nutr. 2011 May;6(2):109-15 [21484164.001]
  • [Cites] J Agric Food Chem. 2000 Jul;48(7):2848-52 [10898634.001]
  • [Cites] Mol Cell Biochem. 2005 May;273(1-2):109-16 [16013445.001]
  • [Cites] Mutat Res. 2004 Nov 2;555(1-2):3-19 [15476848.001]
  • [Cites] Biomed Sci Instrum. 2015;51:31-9 [25996696.001]
  • [Cites] Cancer Res Treat. 2004 Oct;36(5):315-23 [20368822.001]
  • [Cites] Enzymes. 2014;36:195-221 [27102705.001]
  • [Cites] Crit Rev Food Sci Nutr. 2005;45(4):287-306 [16047496.001]
  • [Cites] J Obstet Gynaecol Res. 2006 Apr;32(2):148-54 [16594917.001]
  • [Cites] Anticancer Res. 2010 Jul;30(7):2519-23 [20682977.001]
  • (PMID = 28504421.001).
  • [ISSN] 1752-8062
  • [Journal-full-title] Clinical and translational science
  • [ISO-abbreviation] Clin Transl Sci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


6. Guo Y, Zhi F, Chen P, Zhao K, Xiang H, Mao Q, Wang X, Zhang X: Green tea and the risk of prostate cancer: A systematic review and meta-analysis. Medicine (Baltimore); 2017 Mar;96(13):e6426
Hazardous Substances Data Bank. Green tea .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea and the risk of prostate cancer: A systematic review and meta-analysis.
  • Among these foods, green tea is considered as effective prevention for various cancers.
  • However, clinical trials and previous meta-analyses on the relationship between green tea consumption and the risk of PCa have produced inconsistent outcomes.
  • This study aims to determine the dose-response association of green tea intake with PCa risk and the preventive effect of green tea catechins on PCa risk.
  • Dose-response relations were evaluated with categories of green tea intake.
  • Although there was no statistical significance in the comparison of the highest versus lowest category, there was a trend of reduced incidence of PCa with each 1 cup/day increase of green tea (P = 0.08).
  • Our dose-response meta-analysis further demonstrated that higher green tea consumption was linearly associated with a reduced risk of PCa with more than 7 cups/day.
  • In addition, green tea catechins were effective for preventing PCa with an RR of 0.38 (P = 0.02).
  • In conclusion, our dose-response meta-analysis evaluated the association of green tea intake with PCa risk systematically and quantitatively.
  • And this is the first meta-analysis of green tea catechins consumption and PCa incidence.
  • Our novel data demonstrated that higher green tea consumption was linearly reduced PCa risk with more than 7 cups/day and green tea catechins were effective for preventing PCa.
  • [MeSH-major] Carcinoma / prevention & control. Catechin / therapeutic use. Prostatic Neoplasms / prevention & control. Tea

  • Genetic Alliance. consumer health - Prostate cancer.
  • MedlinePlus Health Information. consumer health - Prostate Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] J Biol Chem. 2003 Mar 28;278(13):11590-600 [12551933.001]
  • [Cites] Cancer Res. 2006 Jan 15;66(2):1234-40 [16424063.001]
  • [Cites] Environ Health Perspect. 1990 Jul;87:173-8 [2269223.001]
  • [Cites] Oncogene. 2008 Mar 27;27(14):2055-63 [17998943.001]
  • [Cites] Toxicology. 2009 Jun 16;260(1-3):28-36 [19464566.001]
  • [Cites] Cancer Causes Control. 2004 Nov;15(9):911-20 [15577293.001]
  • [Cites] Medicine (Baltimore). 2015 Aug;94(33):e1260 [26287411.001]
  • [Cites] Endocrinology. 2000 Mar;141(3):980-7 [10698173.001]
  • [Cites] Crit Rev Food Sci Nutr. 2015;55(6):792-805 [24915354.001]
  • [Cites] Mol Pharmacol. 2002 Oct;62(4):765-71 [12237322.001]
  • [Cites] Nat Clin Pract Oncol. 2005 May;2(5):255-61 [16264961.001]
  • [Cites] Int J Mol Epidemiol Genet. 2010;1(2):114-123 [21191472.001]
  • [Cites] Eur Urol. 2008 Aug;54(2):472-3 [18406041.001]
  • [Cites] Cancer Sci. 2004 Mar;95(3):238-42 [15016323.001]
  • [Cites] Am J Epidemiol. 2012 Jan 1;175(1):66-73 [22135359.001]
  • [Cites] Cancer Causes Control. 2012 Oct;23(10):1635-41 [22864870.001]
  • [Cites] Int J Cancer. 2004 Dec 10;112(5):787-92 [15386386.001]
  • [Cites] Am J Epidemiol. 2008 Jan 1;167(1):71-7 [17906295.001]
  • [Cites] Nutr Cancer. 2011;63(5):663-72 [21667398.001]
  • [Cites] Biochem Biophys Res Commun. 1995 Sep 25;214(3):833-8 [7575552.001]
  • [Cites] Carcinogenesis. 2006 Apr;27(4):833-9 [16387739.001]
  • [Cites] Maturitas. 2012 Dec;73(4):280-7 [22986087.001]
  • [Cites] Epidemiol Rev. 1987;9:1-30 [3678409.001]
  • [Cites] Cancer Prev Res (Phila). 2015 Oct;8(10 ):879-87 [25873370.001]
  • [Cites] World J Surg Oncol. 2014 Feb 14;12:38 [24528523.001]
  • [Cites] Cancer Lett. 2009 Mar 8;275(1):86-92 [18977589.001]
  • [Cites] Oncogene. 2000 Apr 6;19(15):1924-32 [10773882.001]
  • [Cites] Cancer Lett. 1995 Sep 25;96(2):239-43 [7585463.001]
  • [Cites] CA Cancer J Clin. 2015 Mar;65(2):87-108 [25651787.001]
  • [Cites] Pak J Pharm Sci. 2008 Jul;21(3):249-54 [18614420.001]
  • [Cites] Am J Epidemiol. 1992 Jun 1;135(11):1301-9 [1626547.001]
  • [Cites] Int J Cancer. 2004 Dec 10;112(5):823-9 [15386368.001]
  • [Cites] Prostate. 2009 Feb 1;69(2):219-24 [18942120.001]
  • [Cites] J Agric Food Chem. 2008 Oct 8;56(19):9225-9 [18778031.001]
  • [Cites] Cancer Res. 2004 Dec 1;64(23):8715-22 [15574782.001]
  • [Cites] Crit Rev Food Sci Nutr. 2003;43(1):89-143 [12587987.001]
  • [Cites] Int J Cancer. 2004 Jan 1;108(1):130-5 [14618627.001]
  • [Cites] BMC Cancer. 2014 Mar 17;14:197 [24636229.001]
  • [Cites] Cancer Res. 2000 Jan 1;60(1):28-34 [10646846.001]
  • [Cites] Prostate. 2007 Oct 1;67(14):1576-89 [17705241.001]
  • [Cites] Br J Cancer. 2006 Aug 7;95(3):371-3 [16804523.001]
  • [Cites] Asia Pac J Clin Nutr. 2007;16 Suppl 1:453-7 [17392149.001]
  • [Cites] Int J Clin Exp Med. 2014 Nov 15;7(11):3881-91 [25550896.001]
  • [Cites] J Zhejiang Univ Sci B. 2009 Jun;10(6):411-21 [19489106.001]
  • (PMID = 28353571.001).
  • [ISSN] 1536-5964
  • [Journal-full-title] Medicine
  • [ISO-abbreviation] Medicine (Baltimore)
  • [Language] eng
  • [Publication-type] Journal Article; Meta-Analysis; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Tea; 8R1V1STN48 / Catechin
  •  go-up   go-down


7. Singhal K, Raj N, Gupta K, Singh S: Probable benefits of green tea with genetic implications. J Oral Maxillofac Pathol; 2017 Jan-Apr;21(1):107-114

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Probable benefits of green tea with genetic implications.
  • Tea is produced from the <i>Camellia sinensis</i> plant and can generally be divided into categories based on how they are processed.
  • In general, green tea that is unfermented <i>C. sinensis</i> has been considered superior to black tea in health benefits.
  • Oral cavity oxidative stress and inflammation, consequent cigarettes due to nicotine and acrolein, may be reduced in the presence of green tea polyphenols.
  • In addition, green tea polyphenols can close down halitosis through modification of odorant sulfur components.
  • Usually, green tea defends healthy cells from malignant transformation and locally has the ability to induce apoptosis in oral cancer cells.
  • In unison, there is an increasing implication in the health benefits of green tea in the field of oral health.
  • This review will cover recent findings on the therapeutic properties and anticancer health benefits of green tea.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] J Am Coll Nutr. 2006 Apr;25(2):79-99 [16582024.001]
  • [Cites] Eur J Oral Sci. 2010 Apr;118(2):145-50 [20487003.001]
  • [Cites] Food Chem Toxicol. 1997 Aug;35(8):827-33 [9350228.001]
  • [Cites] J Periodontol. 2009 Mar;80(3):372-7 [19254120.001]
  • [Cites] Br J Nutr. 2009 Dec;102(12):1790-802 [19751534.001]
  • [Cites] Molecules. 2007 May 03;12(5):946-57 [17873830.001]
  • [Cites] J Altern Complement Med. 2005 Jun;11(3):521-8 [15992239.001]
  • [Cites] Eur J Clin Nutr. 1996 Jan;50(1):28-32 [8617188.001]
  • [Cites] Nutrition. 2002 May;18(5):443-4 [11985958.001]
  • [Cites] Caries Res. 1999 Nov-Dec;33(6):441-5 [10529529.001]
  • [Cites] Arch Oral Biol. 2012 May;57(5):429-35 [22226360.001]
  • [Cites] J Periodontal Res. 2002 Dec;37(6):433-8 [12472837.001]
  • [Cites] Anticancer Agents Med Chem. 2006 Sep;6(5):389-406 [17017850.001]
  • [Cites] J Periodontal Res. 2004 Oct;39(5):300-7 [15324350.001]
  • [Cites] Proc Natl Acad Sci U S A. 1999 Apr 13;96(8):4524-9 [10200295.001]
  • [Cites] Biol Psychiatry. 2007 Dec 15;62(12):1353-62 [17624318.001]
  • [Cites] J Periodontol. 1993 Jul;64(7):630-6 [8396176.001]
  • [Cites] Am J Clin Nutr. 2007 Oct;86(4):1243-7 [17921409.001]
  • [Cites] BMC Complement Altern Med. 2014 Aug 30;14:322 [25175005.001]
  • [Cites] Arch Intern Med. 2003 Jun 23;163(12):1448-53 [12824094.001]
  • [Cites] Antiviral Res. 2003 Apr;58(2):167-73 [12742577.001]
  • [Cites] J Indian Soc Periodontol. 2012 Apr;16(2):161-7 [23055579.001]
  • [Cites] J Cell Biochem. 2001;82(3):387-98 [11500915.001]
  • [Cites] Cancer Lett. 1998 Jul 17;129(2):173-9 [9719459.001]
  • [Cites] Arch Biochem Biophys. 2000 Apr 15;376(2):338-46 [10775421.001]
  • [Cites] Int J Cardiol. 2006 Apr 14;108(3):301-8 [15978686.001]
  • [Cites] J Periodontal Res. 2010 Feb;45(1):23-30 [19602116.001]
  • [Cites] Explore (NY). 2006 Nov-Dec;2(6):531-9 [17113495.001]
  • [Cites] Proc Natl Acad Sci U S A. 2003 May 13;100(10):6009-14 [12719524.001]
  • [Cites] J Expo Sci Environ Epidemiol. 2008 Mar;18(2):158-66 [17410113.001]
  • [Cites] Oral Microbiol Immunol. 2004 Apr;19(2):118-20 [14871352.001]
  • [Cites] Biochem Pharmacol. 1999 Sep 15;58(6):911-5 [10509743.001]
  • [Cites] Biochem Biophys Res Commun. 2002 Mar 22;292(1):94-101 [11890677.001]
  • [Cites] Biosci Biotechnol Biochem. 1996 May;60(5):745-9 [8704303.001]
  • [Cites] J Med Chem. 2007 Jan 25;50(2):264-71 [17228868.001]
  • [Cites] Rev Saude Publica. 2004 Feb;38(1):100-5 [14963548.001]
  • [Cites] Antimicrob Agents Chemother. 2004 Jun;48(6):1968-73 [15155186.001]
  • [Cites] Antiviral Res. 1993 Aug;21(4):289-99 [8215301.001]
  • [Cites] Ann N Y Acad Sci. 2001 Apr;928:274-80 [11795518.001]
  • [Cites] J Oral Pathol Med. 2007 Nov;36(10):588-93 [17944751.001]
  • [Cites] Ann Nutr Metab. 2004;48(2):95-102 [14988639.001]
  • [Cites] Clin Nutr. 2006 Oct;25(5):790-6 [16698148.001]
  • [Cites] Proc Soc Exp Biol Med. 1999 Apr;220(4):218-24 [10202392.001]
  • [Cites] J Clin Aesthet Dermatol. 2012 Feb;5(2):34-41 [22468171.001]
  • [Cites] Arch Pharm Res. 1998 Jun;21(3):348-52 [9875456.001]
  • [Cites] Eur J Pharmacol. 2004 Oct 1;500(1-3):177-85 [15464031.001]
  • [Cites] Tob Induc Dis. 2003 Sep 15;1(3):219-26 [19570263.001]
  • [Cites] Comp Biochem Physiol C Toxicol Pharmacol. 2001 Feb;128(2):153-64 [11239828.001]
  • [Cites] Heart. 2004 Dec;90(12):1485-6 [15547040.001]
  • [Cites] J Endod. 2010 Jan;36(1):83-6 [20003940.001]
  • [Cites] Drug Metabol Drug Interact. 2004;20(1-2):43-56 [15283302.001]
  • [Cites] J Am Coll Nutr. 2002 Feb;21(1):1-13 [11838881.001]
  • [Cites] Pharmacol Res. 2011 Aug;64(2):105-12 [21624470.001]
  • [Cites] Exp Toxicol Pathol. 2005 Jul;57 Suppl 1:43-73 [16092717.001]
  • [Cites] Iran J Basic Med Sci. 2012 May;15(3):880-4 [23493325.001]
  • [Cites] J Indian Soc Periodontol. 2011 Jan;15(1):18-22 [21772716.001]
  • [Cites] J Nutr Biochem. 2004 Sep;15(9):506-16 [15350981.001]
  • [Cites] J Immunol. 2003 Apr 15;170(8):4335-41 [12682270.001]
  • [Cites] Phytother Res. 2004 Aug;18(8):624-7 [15476306.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2004 Jan;13(1):132-7 [14744744.001]
  • [Cites] J Agric Food Chem. 2004 Mar 24;52(6):1688-92 [15030231.001]
  • [Cites] Toxicol Sci. 2006 Feb;89(2):547-53 [16280382.001]
  • (PMID = 28479696.001).
  • [ISSN] 0973-029X
  • [Journal-full-title] Journal of oral and maxillofacial pathology : JOMFP
  • [ISO-abbreviation] J Oral Maxillofac Pathol
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] India
  • [Keywords] NOTNLM ; Antioxidants / Camellia sinensis / cancer / tea
  •  go-up   go-down


8. Jacob SA, Khan TM, Lee LH: The Effect of Green Tea Consumption on Prostate Cancer Risk and Progression: A Systematic Review. Nutr Cancer; 2017 Apr;69(3):353-364

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The Effect of Green Tea Consumption on Prostate Cancer Risk and Progression: A Systematic Review.
  • This systematic review aimed to assess the clinical benefits of green tea consumption on the progression and prevention of prostate cancer (PCa).
  • Studies must report on the effect of green tea consumption on PCa.
  • A total of 15 articles were included, with 11 reporting on the effect of green tea consumption on PCa prevention, and four reporting on the effect of green tea on treatment.
  • Findings demonstrate that green tea appears to be an effective chemopreventive agent, particularly in those with high-grade prostate intraepithelial neoplasia.
  • Given the limitations in current studies, more well-designed RCTs should be undertaken to determine if green tea indeed has a role in the prevention and treatment of PCa.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 28287319.001).
  • [ISSN] 1532-7914
  • [Journal-full-title] Nutrition and cancer
  • [ISO-abbreviation] Nutr Cancer
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


9. Ni CX, Gong H, Liu Y, Qi Y, Jiang CL, Zhang JP: Green Tea Consumption and the Risk of Liver Cancer: A Meta-Analysis. Nutr Cancer; 2017 Feb-Mar;69(2):211-220

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea Consumption and the Risk of Liver Cancer: A Meta-Analysis.
  • : Green tea is a commonly consumed beverage in Asia and has been suggested to have anticarcinogenic properties.
  • To date, epidemiological evidence of the effect of green tea consumption on liver cancer risk remains ambiguous.
  • The aim of this meta-analysis is to evaluate the association between green tea consumption and the risk of liver cancer.
  • The summary relative risk for the highest consumption (≥5 cups/day) of green tea on liver cancer incidence compared with nondrinkers was 0.62 (95% confidence interval: 0.49-0.79).
  • We also found a trend that the incidence of liver cancer was reduced with the increasing years of green tea intake (significance at >20 yr).
  • A significant dose-response association was found between green tea drinking and liver cancer risk.
  • The downward trend was most obvious when the consumption of green tea increased up to about 4 cups/day.
  • The results showed that the increasing green tea intake may have a preventive effect against liver cancer.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 28095030.001).
  • [ISSN] 1532-7914
  • [Journal-full-title] Nutrition and cancer
  • [ISO-abbreviation] Nutr Cancer
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


10. Shin CM, Lee DH, Seo AY, Lee HJ, Kim SB, Son WC, Kim YK, Lee SJ, Park SH, Kim N, Park YS, Yoon H: Green tea extracts for the prevention of metachronous colorectal polyps among patients who underwent endoscopic removal of colorectal adenomas: A randomized clinical trial. Clin Nutr; 2017 Jan 29;
NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea extracts for the prevention of metachronous colorectal polyps among patients who underwent endoscopic removal of colorectal adenomas: A randomized clinical trial.
  • OBJECTIVES: To determine the preventive effect of green tea extract (GTE) supplements on metachronous colorectal adenoma and cancer in the Korean population.
  • However, there were no significant differences between the 2 groups in terms of body mass index, dietary intakes, serum lipid profiles, fasting serum glucose, and serum C-reactive protein levels (all p > 0.05).

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright © 2017 Elsevier Ltd and European Society for Clinical Nutrition and Metabolism. All rights reserved.
  • (PMID = 28209333.001).
  • [ISSN] 1532-1983
  • [Journal-full-title] Clinical nutrition (Edinburgh, Scotland)
  • [ISO-abbreviation] Clin Nutr
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Keywords] NOTNLM ; Colon polyps / Epigallocatechin gallate / Green tea / Recurrence risk
  •  go-up   go-down






Advertisement