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Items 1 to 10 of about 4898
1. Kim TE, Ha N, Kim Y, Kim H, Lee JW, Jeon JY, Kim MG: Effect of epigallocatechin-3-gallate, major ingredient of green tea, on the pharmacokinetics of rosuvastatin in healthy volunteers. Drug Des Devel Ther; 2017;11:1409-1416

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Effect of epigallocatechin-3-gallate, major ingredient of green tea, on the pharmacokinetics of rosuvastatin in healthy volunteers.
  • Previous in vitro studies have demonstrated the inhibitory effect of green tea on drug transporters.
  • Because rosuvastatin, a lipid-lowering drug widely used for the prevention of cardiovascular events, is a substrate for many drug transporters, there is a possibility that there is interaction between green tea and rosuvastatin.
  • The aim of this study was to investigate the effect of green tea on the pharmacokinetics of rosuvastatin in healthy volunteers.
  • After a 3-day washout period, the subjects received 20 mg of rosuvastatin plus 300 mg of epigallocatechin-3-gallate (EGCG), a major ingredient of green tea (Day 4).

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  • (PMID = 28533679.001).
  • [ISSN] 1177-8881
  • [Journal-full-title] Drug design, development and therapy
  • [ISO-abbreviation] Drug Des Devel Ther
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] New Zealand
  • [Keywords] NOTNLM ; EGCG / drug interaction / drug transporter / green tea / pharmacokinetics / rosuvastatin
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2. Zhao LG, Li HL, Sun JW, Yang Y, Ma X, Shu XO, Zheng W, Xiang YB: Green tea consumption and cause-specific mortality: Results from two prospective cohort studies in China. J Epidemiol; 2017 Jan;27(1):36-41
Hazardous Substances Data Bank. Green tea .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea consumption and cause-specific mortality: Results from two prospective cohort studies in China.
  • BACKGROUND: Green tea is one of the most widely consumed beverages in Asia.
  • While a possible protective role of green tea against various chronic diseases has been suggested in experimental studies, evidence from human studies remains controversial.
  • Hazard ratios (HR) and 95% confidence intervals (CI) for risk of all-cause and cause-specific mortality associated with green tea intake were estimated using Cox proportional hazards regression models.
  • We found that green tea consumption was inversely associated with risk of all-cause mortality (HR 0.95; 95% CI, 0.90-1.01), particularly among never-smokers (HR 0.89; 95% CI, 0.82-0.96).
  • No significant association was observed between green tea intake and cancer mortality (HR 1.01; 95% CI, 0.93-1.10).
  • CONCLUSIONS: Green tea consumption may be inversely associated with risk of all-cause and CVD mortality in middle-aged and elderly Chinese adults, especially among never smokers.
  • [MeSH-major] Cause of Death. Tea

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  • [Copyright] Copyright © 2016 The Authors. Production and hosting by Elsevier B.V. All rights reserved.
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  • (PMID = 28135196.001).
  • [ISSN] 1349-9092
  • [Journal-full-title] Journal of epidemiology
  • [ISO-abbreviation] J Epidemiol
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / UM1 CA173640; United States / NCI NIH HHS / CA / UM1 CA182910
  • [Publication-type] Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Tea
  • [Keywords] NOTNLM ; All-cause mortality / CVD mortality / Cancer mortality / Cohort study / Green tea
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3. Allam AA, Gabr SA, Ajarem J, Alghadir AH, Sekar R, Chow BK: GENO PROTECTIVE AND ANTI-APOPTOTIC EFFECT OF GREEN TEA AGAINST PERINATAL LIPOPOLYSACCHARIDE-EXPOSURE INDUCED LIVER TOXICITY IN RAT NEWBORNS. Afr J Tradit Complement Altern Med; 2017;14(2):166-176

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] GENO PROTECTIVE AND ANTI-APOPTOTIC EFFECT OF GREEN TEA AGAINST PERINATAL LIPOPOLYSACCHARIDE-EXPOSURE INDUCED LIVER TOXICITY IN RAT NEWBORNS.
  • BACKGROUND: This study aims to examine the protective effect of green tea on the disturbances in oxidative stress and apoptosis related factors, mostly produced due to perinatal lipopolysaccharide (LPS) exposure, that subsequently induces liver cell damage.
  • MATERIALS AND METHODS: Anti-free radical, Antioxidant, scavenging, geno-protective, and antiapoptotic activity of aqueous green tea extract (AGTE) were assessed against LPS-induced hepatic dysfunction in newborn-rats.

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  • (PMID = 28573233.001).
  • [ISSN] 2505-0044
  • [Journal-full-title] African journal of traditional, complementary, and alternative medicines : AJTCAM
  • [ISO-abbreviation] Afr J Tradit Complement Altern Med
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Nigeria
  • [Keywords] NOTNLM ; apoptosis / green tea / lipopolysaccharide / liver dysfunction / newborns
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4. Liu SM, Ou SY, Huang HH: Green tea polyphenols induce cell death in breast cancer MCF-7 cells through induction of cell cycle arrest and mitochondrial-mediated apoptosis. J Zhejiang Univ Sci B; 2017 Feb.;18(2):89-98
Hazardous Substances Data Bank. Green tea .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea polyphenols induce cell death in breast cancer MCF-7 cells through induction of cell cycle arrest and mitochondrial-mediated apoptosis.
  • In order to study the molecular mechanisms of green tea polyphenols (GTPs) in treatment or prevention of breast cancer, the cytotoxic effects of GTPs on five human cell lines (MCF-7, A549, Hela, PC3, and HepG2 cells) were determined and the antitumor mechanisms of GTPs in MCF-7 cells were analyzed.
  • [MeSH-major] Apoptosis. Cell Cycle Checkpoints / drug effects. Mitochondria / metabolism. Polyphenols / pharmacology
  • [MeSH-minor] A549 Cells. Caspase 3 / metabolism. Caspase 9 / metabolism. Cell Line, Tumor. Cell Survival. Chromatin / chemistry. DNA Fragmentation. Flow Cytometry. Guanosine Triphosphate / metabolism. HeLa Cells. Hep G2 Cells. Humans. MCF-7 Cells. Membrane Potential, Mitochondrial. Reactive Oxygen Species / metabolism. Tea

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  • (PMID = 28124838.001).
  • [ISSN] 1862-1783
  • [Journal-full-title] Journal of Zhejiang University. Science. B
  • [ISO-abbreviation] J Zhejiang Univ Sci B
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Chromatin; 0 / Polyphenols; 0 / Reactive Oxygen Species; 0 / Tea; 86-01-1 / Guanosine Triphosphate; EC 3.4.22.- / CASP3 protein, human; EC 3.4.22.- / CASP9 protein, human; EC 3.4.22.- / Caspase 3; EC 3.4.22.- / Caspase 9
  • [Keywords] NOTNLM ; Green tea polyphenol (GTP); Breast cancer; MCF-7 cells; Mitochondrial-mediated apoptosis; Cell death; Cell cycle arrest
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5. Guo Y, Zhi F, Chen P, Zhao K, Xiang H, Mao Q, Wang X, Zhang X: Green tea and the risk of prostate cancer: A systematic review and meta-analysis. Medicine (Baltimore); 2017 Mar;96(13):e6426
Hazardous Substances Data Bank. Green tea .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea and the risk of prostate cancer: A systematic review and meta-analysis.
  • Among these foods, green tea is considered as effective prevention for various cancers.
  • However, clinical trials and previous meta-analyses on the relationship between green tea consumption and the risk of PCa have produced inconsistent outcomes.
  • This study aims to determine the dose-response association of green tea intake with PCa risk and the preventive effect of green tea catechins on PCa risk.
  • Dose-response relations were evaluated with categories of green tea intake.
  • Although there was no statistical significance in the comparison of the highest versus lowest category, there was a trend of reduced incidence of PCa with each 1 cup/day increase of green tea (P = 0.08).
  • Our dose-response meta-analysis further demonstrated that higher green tea consumption was linearly associated with a reduced risk of PCa with more than 7 cups/day.
  • In addition, green tea catechins were effective for preventing PCa with an RR of 0.38 (P = 0.02).
  • In conclusion, our dose-response meta-analysis evaluated the association of green tea intake with PCa risk systematically and quantitatively.
  • And this is the first meta-analysis of green tea catechins consumption and PCa incidence.
  • Our novel data demonstrated that higher green tea consumption was linearly reduced PCa risk with more than 7 cups/day and green tea catechins were effective for preventing PCa.
  • [MeSH-major] Carcinoma / prevention & control. Catechin / therapeutic use. Prostatic Neoplasms / prevention & control. Tea

  • Genetic Alliance. consumer health - Prostate cancer.
  • MedlinePlus Health Information. consumer health - Prostate Cancer.
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  • (PMID = 28353571.001).
  • [ISSN] 1536-5964
  • [Journal-full-title] Medicine
  • [ISO-abbreviation] Medicine (Baltimore)
  • [Language] eng
  • [Publication-type] Journal Article; Meta-Analysis; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Tea; 8R1V1STN48 / Catechin
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6. Zhou Q, Chen Z, Lee J, Li X, Sun W: Proteomic analysis of tea plants (Camellia sinensis) with purple young shoots during leaf development. PLoS One; 2017;12(5):e0177816

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Proteomic analysis of tea plants (Camellia sinensis) with purple young shoots during leaf development.
  • Tea products made from purple leaves are highly preferred by consumers due to the health benefits.
  • This study developed a proteome reference map related to color changes during leaf growth in tea (Camellia sinensis) plant with purple young shoots using two-dimensional electrophoresis (2-DE).
  • The pronounced changes in the proteomic profile between tender purple leaves (TPL) and mature green leaves (MGL) included:.
  • The higher abundance of glutamine synthetase (GS) proteins related to the theanine biosynthesis may improve the flavor of tea products from TPL materials.

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  • (PMID = 28520776.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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7. Porciani PF, Grandini S: Effect of Green Tea-Added Tablets on Volatile Sulfur-Containing Compounds in the Oral Cavity. J Clin Dent; 2016 Dec;27(4):110-113
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Effect of Green Tea-Added Tablets on Volatile Sulfur-Containing Compounds in the Oral Cavity.
  • OBJECTIVES: A controlled, clinical, double-blind, cross-over study was conducted to assess the efficacy of sugar-free tablets containing green tea extract on oral volatile sulfur-containing compounds (VSC) versus placebo tablets for 30 minutes.
  • All eligible participants had to avoid professional oral hygiene and drugs for two weeks, to not be menstruating, to avoid brushing their teeth and tongue, to not smoke, to not consume alcohol, coffee or tea, nor onion, garlic, or licorice for six hours before the test.
  • The test tablet (0.7 g) contained 0.05% green tea extract (equivalent of 1 mg polyphenols for three tablets); the control tablet was identical but without the active agent.
  • None reported problems linked to green tea.
  • CONCLUSIONS: Tablets containing green tea extract can statistically significantly reduce the oral VSC levels immediately, and after 30 minutes.

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  • (PMID = 28391664.001).
  • [ISSN] 0895-8831
  • [Journal-full-title] The Journal of clinical dentistry
  • [ISO-abbreviation] J Clin Dent
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; green tea / halitosis / oral malodor / volatile sulfur compounds
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8. Azimi S, Mansouri Z, Bakhtiari S, Tennant M, Kruger E, Rajabibazl M, Daraei A: Does green tea consumption improve the salivary antioxidant status of smokers? Arch Oral Biol; 2017 Jun;78:1-5

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Does green tea consumption improve the salivary antioxidant status of smokers?
  • OBJECTIVE: 
Considering the higher rate of oral cancer, and reduction in salivary antioxidants in smokers as indicated in previous studies, antioxidant- containing nutrients such as green tea, seem to be beneficial in counteracting against oxidative stress in this group.
  • This study assessed the salivary total antioxidant alteration in smokers compared to nonsmokers, after short-tem (7days) and long-term (3 weeks), green tea drinking.
  • Participants were instructed to drink two cups of green tea per day, by dissolving 2g of green tea in 150ml of hot water for each cup.
  • There was also a significant difference between two groups in salivary total antioxidant capacity after one week and three weeks of green tea consumption (P<0.001).
  • However, there was an upward trend in both smokers and non-smokers over the study period (after tea drinking).
  • CONCLUSIONS: Results support the effectiveness of green tea consumption in salivary antioxidants enhancement in smokers, in both the short- and long term.

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  • [Copyright] Copyright © 2017 Elsevier Ltd. All rights reserved.
  • (PMID = 28189030.001).
  • [ISSN] 1879-1506
  • [Journal-full-title] Archives of oral biology
  • [ISO-abbreviation] Arch. Oral Biol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Keywords] NOTNLM ; Antioxidant capacity / Green tea / Saliva / Smokers
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9. Li J, Sapper TN, Mah E, Moller MV, Kim JB, Chitchumroonchokchai C, McDonald JD, Bruno RS: Green tea extract treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by lowering TNFR1 and TLR4 expression and ligand availability. J Nutr Biochem; 2017 Mar;41:34-41
NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green tea extract treatment reduces NFκB activation in mice with diet-induced nonalcoholic steatohepatitis by lowering TNFR1 and TLR4 expression and ligand availability.
  • We hypothesized that antiinflammatory activities of green tea extract (GTE) during NASH would lower tumor necrosis factor receptor-1 (TNFR1)- and Toll-like receptor-4 (TLR4)-mediated NFκB activation.
  • These data suggest that dietary GTE treatment reduces hepatic inflammation in NASH by decreasing proinflammatory signaling through TNFR1 and TLR4 that otherwise increases NFκB activation and liver injury.

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  • [Copyright] Copyright © 2016 Elsevier Inc. All rights reserved.
  • (PMID = 28038359.001).
  • [ISSN] 1873-4847
  • [Journal-full-title] The Journal of nutritional biochemistry
  • [ISO-abbreviation] J. Nutr. Biochem.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Keywords] NOTNLM ; Green tea / Inflammation / NASH / Nonalcoholic steatohepatitis / TLR4 / TNFR1
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10. Maiti S, Acharyya N, Ghosh TK, Ali SS, Manna E, Nazmeen A, Sinha NK: Green Tea (Camellia sinensis) Protects Against Arsenic Neurotoxicity via Antioxidative Mechanism And Activation of Superoxide Dismutase Activity. Cent Nerv Syst Agents Med Chem; 2017 Feb 01;

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Green Tea (Camellia sinensis) Protects Against Arsenic Neurotoxicity via Antioxidative Mechanism And Activation of Superoxide Dismutase Activity.
  • Here, the protective role of Green tea (Camellia sinensis or CS; 10mg/ml aqueous) has been evaluated against arsenic-induced (0.6ppm/100g bw/28 days) cerebral/cerebellar tissue degeneration, oxidative-threats and neurotransmitter deregulation in female rats.

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  • (PMID = 28155600.001).
  • [ISSN] 1875-6166
  • [Journal-full-title] Central nervous system agents in medicinal chemistry
  • [ISO-abbreviation] Cent Nerv Syst Agents Med Chem
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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